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Another significant difference between Hoffmann's reflex and the Babinski sign is their mechanism of reflex. Hoffmann's reflex is a deep tendon reflex (spindle fibre) with a monosynaptic reflex pathway in Rexed lamina IX of the spinal cord, normally fully inhibited by descending input. On the other hand, the plantar reflex is more complicated ...
H-reflex is analogous to the mechanically induced spinal stretch reflex (for example, knee jerk reflex). "The primary difference between the H-reflex and the spinal stretch reflex is that the H-reflex bypasses the muscle spindle, and, therefore, is a valuable tool in assessing modulation of monosynaptic reflex activity in the spinal cord."
Hoffmann's sign: Johann Hoffmann: neurology: corticospinal tract lesions: tapping distal phalanx of 3rd or 4th finger elicits flexion of same in thumb Hollenhorst plaque: Robert Hollenhorst: ophthalmology: hypertension, coronary artery disease, and/or diabetes: cholesterol embolus(i) of retinal artery(ies) Homans' sign: John Homans: thrombosis ...
Babinski sign. The irritative phenomena are present if there is visible flection of the thumb, which goes to opposition: Hoffmann's sign – The patient's middle finger is flicked from the nail side down using the examiners index finger. Tromner's sign - The patient's middle finger is flicked from underneath using the examiner's index finger.
Hering–Breuer reflex — is a reflex triggered to prevent over-inflation of the lung; Hoffmann's reflex — also known as the finger flexor reflex; middle finger and thumb response. Test can indicate both neurological damage and nerve regeneration; often combined with the Babinski reflex test. Jaw jerk reflex
Lhermitte's sign may also appear during or following high-dose chemotherapy. [7] [8] Irradiation of the cervical spine may also evoke it as an early delayed radiation injury, which occurs within 4 months of radiation therapy. [citation needed] Delayed onset Lhermitte's sign has been reported following head and/or neck trauma.
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As the brain matures, certain areas (usually within the frontal lobes) exert an inhibitory effect, thus causing the reflex to disappear. When disease processes disrupt these inhibitory pathways, the reflex is "released" from inhibition and can be elicited once again, hence the term "frontal release sign".