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The peak mitral annular velocity during early filling, e' is a measure of left ventricular diastolic function, and has been shown to be relatively independent of left ventricular filling pressure. [ 12 ] [ 13 ] [ 14 ] If there is impaired relaxation ( Diastolic dysfunction ), the e' velocity decreases.
The mean velocity in the aorta varies over the cardiac cycle. During systole the mean velocity rises to a peak, then it falls during diastole. This pattern is repeated with each squeezing pulse of the heart. The highest velocities are found at the exit of the valve during systole.
In ultrasound it is usually measured from the velocity gradient SR = (v 2 - v 1)/L where v 2 and v 1 are the myocardial velocities at two different points, and L is the instantaneous distance between them. This is thus equivalent to the velocity difference per length unit (the spatial derivative of velocity) and has the unit s −1. Strain is ...
One parameter to quantify this difference is the pulsatility index (PI), which is equal to the difference between the peak systolic velocity and the minimum diastolic velocity divided by the mean velocity during the cardiac cycle. This value decreases with distance from the heart. [20]
Applying spectral Doppler to the renal artery and selected interlobular arteries, peak systolic velocities, resistive index, and acceleration curves can be estimated (Figure 4) (e.g., peak systolic velocity of the renal artery above 180 cm/s is a predictor of renal artery stenosis of more than 60%, and a resistive index, which is a calculated ...
In PC-MRI, the quantitative analysis of stroke volume, mean peak velocity, and peak systolic velocity is possible only in the plane that is perpendicular to the unidirectional flow. Additionally, it is not possible to calculate multidirectional flow in multiaxial planes in 2D or 3D PC-MRI.
The E/A ratio is a marker of the function of the left ventricle of the heart. It represents the ratio of peak velocity blood flow from left ventricular relaxation in early diastole (the E wave) to peak velocity flow in late diastole caused by atrial contraction (the A wave). [1]
The slope of ESPVR (Ees) represents the end-systolic elastance, which provides an index of myocardial contractility. The ESPVR is relatively insensitive to changes in preload, afterload, and heart rate. This makes it an improved index of systolic function over other hemodynamic parameters like ejection fraction, cardiac output, and stroke volume.