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Paroxysmal supraventricular tachycardia (PSVT) is a type of supraventricular tachycardia, named for its intermittent episodes of abrupt onset and termination. [3] [6] Often people have no symptoms. [1] Otherwise symptoms may include palpitations, feeling lightheaded, sweating, shortness of breath, and chest pain. [2] The cause is not known. [3]
There are four main types of SVT: atrial fibrillation, atrial flutter, paroxysmal supraventricular tachycardia (PSVT), and Wolff–Parkinson–White syndrome. [2] The symptoms of SVT include palpitations, feeling of faintness, sweating, shortness of breath, and/or chest pain. [1]
Prevent recurrence of paroxysmal supraventricular tachycardia; Reduce ventricular rate in patients with atrial fibrillation; V Adenosine; Digoxin; Magnesium sulfate; Work by other or unknown mechanisms Contraindicated in ventricular arrhythmias; Adenosine is used to treat supraventricular tachycardias, especially in heart failure and atrial ...
Supraventricular tachycardia (SVT) is an umbrella term for fast heart rhythms arising from the upper part of the heart. [4] There are four main types of SVT: atrial fibrillation , atrial flutter , paroxysmal supraventricular tachycardia (PSVT), and Wolff–Parkinson–White syndrome . [ 4 ]
Vagal maneuvers are the first-line treatment of hemodynamically stable supraventricular tachycardia, serving to slow down or terminate the arrhythmia. [3] Vagal maneuvers have a reported success rate of conversion to sinus rhythm for SVT around 20-40%, possibly being higher for AVNRT (an SVT associated with a bypass tract). [ 4 ]
Verapamil is used for controlling ventricular rate in supraventricular tachycardia (SVT) and migraine headache prevention. [17]Verapamil is also used for the treatment of angina (chronic stable, vasospastic or Prinzmetal variant), unstable angina (crescendo, preinfarction), and for the prevention of paroxysmal supraventricular tachycardia (PSVT).
The Valsalva maneuver may be used to arrest episodes of supraventricular tachycardia. [12] [13] Blood pressure (BP) rises at onset of straining-because the increased intrathoracic pressure (ITP) is added to the pressure in the aorta. It then falls because the ITP compresses the veins, decreasing the venous return and cardiac output.
Sinus node dysfunction can also present with sudden sinus arrest with or without junctional escape, sinoatrial block, prolonged asystolic period followed by tachycardias, or tachycardia-bradycardia syndrome presenting as various atrial arrhythmias such as atrial fibrillation, flutter, tachycardia, or paroxysmal supraventricular tachycardia. [7] [5]