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Bufotenin, also known as dimethylserotonin or as 5-hydroxy-N,N-dimethyltryptamine (5-HO-DMT), is a tryptamine derivative, more specifically, a dimethyltryptamine (DMT) analogue, related to the neurotransmitter serotonin. It is an alkaloid found in some species of mushrooms, plants and toads, especially the skin.
As with DMT, CYB004 is a potent agonist of the serotonin 5-HT 2A receptor and produces psychedelic-like effects in animals. [ 1 ] [ 5 ] [ 3 ] However, CYB004, due to its deuteration, is more resistant to metabolism than DMT and shows a longer elimination half-life (by 2.5- to 2.9-fold) and slower clearance (by 38 to 55%) in animals. [ 3 ]
[5] [1] [4] Like DMT, 5-MeO-DMT has a very rapid onset of action and short duration. [1] [4] However, 5-MeO-DMT is 4- to 10-fold more potent than DMT in humans. [2] 5-MeO-DMT was first synthesized in 1936 by Toshio Hoshino [9] , professor of Tokyo Institute of Technology, and was first isolated from a natural source in 1959. [5]
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[1] [5] [4] However, its affinities for these receptors are lower than those of psilocin (by 8-, 6-, and 26-fold, respectively). [1] [5] [4] Additionally, in another study, the EC 50 Tooltip half-maximal effective concentration value of 4-HO-TMT in activating the serotonin 5-HT 2A receptor was 324-fold lower than that of psilocin (6800 and 21 ...
[1] [3] In contrast to peripherally administered bufotenine, O-acetylbufotenine readily enters the brain in animals and produces robust psychedelic-like effects. [1] [2] In addition, O-acetylbufotenine was more potent than N,N-dimethyltryptamine (DMT) or 5-methoxy-N,N-dimethyltryptamine (5-MeO-DMT; O-methylbufotenine) in animals.
5-Ethoxy-DMT (5-ethoxy-N,N-dimethyltryptamine, 5-EtO-DMT, O-ethylbufotenine) is a tryptamine derivative which has been previously synthesized as a chemical intermediate, but has not been studied to determine its pharmacology.
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