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Vitreous hemorrhage is the extravasation, or leakage, of blood into the areas in and around the vitreous humor of the eye. [1] The vitreous humor is the clear gel ...
The extent and location of the intraocular hemorrhage determine the degree of vision impairment. Because of clots or debris in the vitreous cavity, patients may suffer floaters, which are spots or threads in their field of vision. In extreme situations, extensive vitreous bleeding may result in a total loss of the red reflex.
Intraocular hemorrhage (sometimes called hemophthalmos or hemophthalmia) is bleeding inside the eye (oculus in Latin).Bleeding can occur from any structure of the eye where there is vasculature or blood flow, including the anterior chamber, vitreous cavity, retina, choroid, suprachoroidal space, or optic disc.
The ghost cells develop within the vitreous cavity, 1–3 weeks after vitreous hemorrhage. [1] They obstruct the trabecular meshwork and eventually the pressure inside eye (intraocular pressure) increases and leads to glaucoma. A variety of ocular conditions may cause GCG.
Blood typically exits the wound in spurts, rather than in a steady flow; the blood spurts out in time with the heartbeat. The amount of blood loss can be copious, and can occur very rapidly. [10] Venous bleeding: This blood is flowing from a damaged vein. As a result, it is blackish in colour (due to the lack of oxygen it transports) and flows ...
[1] [2] [3] Emergency treatment of resulting injuries falls under the realm of emergency care and effective patient triage, often incorporating protocols for blunt and penetrating trauma. As a result, physicians have devised a concise algorithm for the treatment of patients with ocular injuries secondary to blast trauma.
In general, branch retinal vein occlusion has a good prognosis: after 1 year 50–60% of eyes have been reported to have a final visual acuity of 20/40 or better even without any treatment. With time the dramatic picture of an acute branch retinal vein occlusion becomes more subtle, hemorrhages fade so that the retina can look almost normal.
Proliferative sickle retinopathy is the most severe ocular complication of sickle cell disease. Even though PSCR begins in the first decade of life, the condition remains asymptomatic and unnoticed until visual symptoms occur due to vitreous hemorrhage or retinal detachment.