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Baroreflex activation is distinct from vagal stimulation. [8] [9] Electrical stimulation of the external surfaces of the carotid sinus activates baroreceptors believed to be in the adventitia of the artery. This stimulates an afferent limb which activates central nervous system pathways that in turn exert two different but synergistic autonomic ...
The carotid sinus baroreceptor can be oversensitive to manual stimulation from the pressure applied at the carotid sinus at the carotid bifurcation. It is a condition known as 'carotid sinus hypersensitivity' (CSH), 'carotid sinus syndrome' or 'carotid sinus syncope', in which manual stimulation causes large changes in heart rate and blood ...
When blood pressure rises, the carotid and aortic sinuses are distended further, resulting in increased stretch and, therefore, a greater degree of activation of the baroreceptors. At normal resting blood pressures, many baroreceptors are actively reporting blood pressure information and the baroreflex is actively modulating autonomic activity.
Reflex bradycardia is a bradycardia (decrease in heart rate) in response to the baroreceptor reflex, one of the body's homeostatic mechanisms for preventing abnormal increases in blood pressure. In the presence of high mean arterial pressure , the baroreceptor reflex produces a reflex bradycardia as a method of decreasing blood pressure by ...
Arterial baroreceptors are located in the carotid sinus (at the bifurcation of common carotid artery into external and internal carotids) and in the aortic arch. [5] The baroreceptors can identify the changes in both the average blood pressure or the rate of change in pressure with each arterial pulse.
High pressure receptors or high pressure baroreceptors are the baroreceptors found within the aortic arch and carotid sinus. They are only sensitive to blood pressures above 60 mmHg . When these receptors are activated they elicit a depressor response; which decreases the heart rate and causes a general vasodilation .
It conveys information from the baroreceptors of the carotid sinus to the vasomotor center in the brainstem (in order to mediate blood pressure homeostasis), and from chemoreceptors of the carotid body [further explanation needed] (mainly conveying information about partial pressures of blood oxygen, and carbon dioxide).
Carotid bodies are considered the primary peripheral chemoreceptor and have been shown to contribute more to a hypoxic response. However, in the chronic absence of the carotid body, the aortic body is able to perform a similar respiratory regulatory role, suggesting that it possesses efficacious mechanisms of signal transduction as well. [5]
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