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Acne (/ˈækni/ ACK-nee), also known as acne vulgaris, is a long-term skin condition that occurs when dead skin cells and oil from the skin clog hair follicles. [10] Typical features of the condition include blackheads or whiteheads , pimples , oily skin, and possible scarring .
Comedo extraction is a widely used method of treatment for acne vulgaris. [1] A dermatologist or cosmetologist may extract blackheads (open comedones) using gentle pressure around the pore opening, and whiteheads (closed comedones) by incision with a large needle or a blade. [2] If performed skillfully, this treatment may be beneficial to the ...
Severe acne usually indicates the necessity of prescription medication to treat the pimples. Prescription medications used to treat acne and pimples include isotretinoin , which is a retinoid , anti-seborrheic medications, anti-androgen medications, hormonal treatments, alpha hydroxy acid , azelaic acid , and keratolytic soaps.
Acne vulgaris Dissecting cellulitis of the scalp Rhinophyma. Acneiform eruptions are caused by changes in the pilosebaceous unit. [27] [28] Acne aestivalis (Mallorca acne) [nb 1] [nb 2] [nb 3] Acne conglobata; Acne cosmetica (cosmetic acne) Acne fulminans (acute febrile ulcerative acne)
Acne vulgaris has a number of different treatments, not all of which are pharmacological in nature. Subcategories This category has the following 2 subcategories, out of 2 total.
Acne necrotica, primary lesions that are pruritic or painful erythematous follicular-based papules that develop central necrosis and crusting and heal with a varioliform scar; Acne rosacea, a red rash predominantly on the face; Acne vulgaris (common acne) Baby acne, a rash seen on the cheeks, chin, and forehead of infants; Blackheads
The partition coefficient (P) of minocycline has been reported to be 39.4 (i.e., log P of 1.60). [72] However, other sources have stated the experimental log P of minocycline to be 0.05, [71] 0.5, [73] and 1.1. [74] In any case, minocycline is consistently described as a highly lipophilic compound with excellent tissue penetration and distribution.
The product of fusB-type resistance genes is a 213-residue cytoplasmic protein which interacts in a 1:1 ratio with EF-G. FusB-type proteins bind in a region distinct from fusidic acid to induce a conformational change which results in liberation of EF-G from the ribosome, allowing the elongation factor to participate in another round of ...