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Large placental infarcts are associated with vascular abnormalities, e.g. hypertrophic decidual vasculopathy, as seen in hypertension. [1] Very large infarcts lead to placental insufficiency and may result in fetal death. Placental infarcts are generally detected after birth, although using ultrasound may be a way to notice infarcts prenatally.
Which trigger pro-coagulator molecules to be released into the blood stream causing action of the coagulator cascade, eventually leading to placental infarction. [5] Risk factors such as diabetes, chronic blood pressure and multiple pregnancies can increase the risk of developing placental disease. [ 3 ]
Histopathology of placenta with increased syncytial knotting of chorionic villi, with two knots pointed out. The following characteristics of placentas have been said to be associated with placental insufficiency, however all of them occur in normal healthy placentas and full term healthy births, so none of them can be used to accurately diagnose placental insufficiency: [citation needed]
It shares symptoms with maternal floor infarction. In mothers who have already experienced it, there is a high risk of recurrence. Recurrence may be around 18%, [11] although in cases where miscarriage occur in the first trimester, it may be as high as 50%. [10]
Velamentous cord insertion impacts fetal development during pregnancy by impairing the development of the placenta [2] and modifying the efficiency of placental function. [17] This can manifest in a range of adverse perinatal outcomes, such as fetal growth restriction, [ 4 ] [ 5 ] placental abruption, [ 3 ] [ 6 ] [ 16 ] [ 18 ] abnormal fetal ...
Intrauterine hypoxia (also known as fetal hypoxia) occurs when the fetus is deprived of an adequate supply of oxygen.It may be due to a variety of reasons such as prolapse or occlusion of the umbilical cord, placental infarction, maternal diabetes (prepregnancy or gestational diabetes) [1] and maternal smoking.
Alkaline phosphatase, placental type is a membrane-bound glycosylated dimeric enzyme, also referred to as the heat-stable form, that is expressed primarily in the placenta, although it is closely related to the intestinal form of the enzyme as well as to the placental-like form. [7]
It takes place in normal pregnancies as well as when there are obstetric or trauma related complications to pregnancy. Normally the maternal circulation and the fetal circulation are kept from direct contact with each other, with gas and nutrient exchange taking place across a membrane in the placenta made of two layers, the syncytiotrophoblast ...