Search results
Results from the WOW.Com Content Network
The 4 substrates of this enzyme are (4-hydroxyphenyl)ethan-1-one, NADPH, H +, and O 2, whereas its 3 products are hydroquinone acetate ester, NADP +, and H 2 O. This enzyme belongs to the family of oxidoreductases , specifically those acting on paired donors, with O2 as oxidant and incorporation or reduction of oxygen.
4-Hydroxyacetophenone monooxygenase is an enzyme that transforms piceol into O-acetylhydroquinone. This enzyme is found in Pseudomonas fluorescens. See also ...
The mechanism of phenol oxidation with hypervalent iodine reagents begins with the formation of an aryloxyiodonium(III) intermediate. Inter- or intramolecular nucleophilic attack then takes place, either in one step or in two via an oxenium ion. [ 4 ]
The mechanism of carbonyl oxidation by iodine(III) reagents varies as a function of substrate structure and reaction conditions, but some generalizations are possible. Under basic conditions, the active iodinating species are iodine(III) compounds in which any relatively acidic ligands on iodine (such as acetate) have been replaced by alkoxide ...
Pages for logged out editors learn more. Contributions; Talk; 4-Hydroxyacetophenone
4-Hydroxyacetophenone (p-hydroxyacetophenone, piceol) Index of chemical compounds with the same name This set index article lists chemical compounds articles associated with the same name.
The iodinium ion is opened via S N 2 reaction by acetic acid (or silver acetate) to give the first intermediate, the iodo-acetate (4). Through anchimeric assistance, the iodine is displaced via another S N 2 reaction to give an oxonium ion (5), which is subsequently hydrolyzed to the give the mono-ester (6). The mechanism of the Woodward cis ...
The mechanism of action of nitisinone involves inhibition of 4-Hydroxyphenylpyruvate dioxygenase (HPPD). [5] [6] This is a treatment for patients with Tyrosinemia type 1 as it prevents the formation of 4-Maleylacetoacetic acid and fumarylacetoacetic acid, which have the potential to be converted to succinyl acetone, a toxin that damages the liver and kidneys. [4]