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Prochlorperazine can also cause a life-threatening condition called neuroleptic malignant syndrome (NMS). Some symptoms of NMS include high fever, stiff muscles, neck muscle spasms, confusion, irregular pulse or blood pressure, fast heart rate (tachycardia), sweating, and abnormal heart rhythms (arrhythmias).
The symptoms can be acute (short-term) or chronic (long-term). They include movement dysfunction such as dystonia (continuous spasms and muscle contractions), akathisia (may manifest as motor restlessness), [ 1 ] parkinsonism characteristic symptoms such as rigidity , bradykinesia (slowness of movement), tremor , and tardive dyskinesia ...
Frequently a result of antiemetics such as the neuroleptics (e.g., prochlorperazine) or metoclopramide. Can be caused by Chlorpromazine. Oromandibular dystonia: muscles of the jaw and muscles of tongue: Causes distortions of the mouth and tongue. Spasmodic dysphonia/Laryngeal dystonia muscles of larynx
Keppra (levetiracetam) – an anticonvulsant drug which is sometimes used as a mood stabilizer and has potential benefits for other psychiatric and neurologic conditions such as Tourette syndrome, anxiety disorder, and Alzheimer's disease; Klonopin – anti-anxiety and anti-epileptic medication of the benzodiazepine class
Other causes can include aromatic L-amino acid decarboxylase deficiency, [9] postencephalitic Parkinson's, Tourette's syndrome, multiple sclerosis, neurosyphilis, head trauma, bilateral thalamic infarction, lesions of the fourth ventricle, cystic glioma of the third ventricle, herpes encephalitis, kernicterus and juvenile Parkinson's disease.
[28] [29] Akathisia can commonly be mistaken for agitation secondary to psychotic symptoms or mood disorder, antipsychotic dysphoria, restless legs syndrome, anxiety, insomnia, drug withdrawal states, tardive dyskinesia, or other neurological and medical conditions. [30] The controversial diagnosis of "pseudoakathisia" is sometimes given. [1]
What causes low testosterone? Many things can lower testosterone, including age, lack of exercise, mental and emotional stress, some medications, and type 2 diabetes .
Serotonin agonists can bind to and activate serotonin receptors, increasing the levels of serotonin in the CNS and resultingly increasing the occurrence of behaviours associated with anxiety. [9] Research supports the resulting anxiogenic effects of agents such as LY-293,284 and mCPP [ 9 ] in the CNS.