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In 1997, ondansetron was the subject of a meta-analysis case study. Researchers examined 84 trials, with 11,980 people receiving ondansetron, published between 1991 and September 1996. Intravenous ondansetron 4 mg versus placebo was investigated in 16 reports and three further reports which had been duplicated a total of six times.
The recognition threshold or arousal threshold of olfactory neurons is the concentration at which you can identify an odor (air, water and fat). The odour activity value is the concentration divided by the threshold. The flavor impact is the value the rate of change in perception with concentration.
“The vagina is self-cleaning…there's no need to do anything that would potentially disrupt the vaginal flora and the normal pH environment of vagina,” explains Dr. King. Basically, you’re ...
Women gave samples of when they were fertile and less fertile. The men in the study would smell and choose which sample between the two they were more attracted to. Women give off a more favorable smell the more fertile they are; in other words, men notice this and choose the more fertile sample rather than the less fertile sample. [49]
The best-known case study involves the synchronization of menstrual cycles among women based on unconscious odor cues, the McClintock effect, named after the primary investigator, Martha McClintock, of the University of Chicago. [7] [8] A group of women were exposed to a whiff of perspiration from other women. Depending on the time in the month ...
Alcohol is a tiny molecule, bathing nearly every cell in the body when we drink. The basic trajectory of liquor in the body is from a person's mouth, through the esophagus, to the stomach ...
Parosmia is a distortion in the perception of an odorant. Odorants smell different from what one remembers. Phantosmia is the perception of an odor when no odorant is present. The cause of dysosmia still remains a theory. It is typically considered a neurological disorder and clinical associations with the disorder have been made. [3]
Alcoholic ketoacidosis is caused by complex physiology that is the result of prolonged and heavy alcohol intake, usually in the setting of poor nutrition. Chronic alcohol use can cause depleted hepatic glycogen stores and ethanol metabolism further impairs gluconeogenesis.