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In the body, homocysteine can be recycled into methionine or converted into cysteine with the aid of vitamin B 6, B 9, and B 12. [3] High levels of homocysteine in the blood (hyperhomocysteinemia) is regarded as a marker of cardiovascular disease, likely working through atherogenesis, which can result in ischemic injury.
B 12 deficiency in pregnant women is increasingly common [40] and has been shown to be associated with major maternal health implications, including increased obesity, [40] higher body mass index (BMI), [41] insulin resistance, [39] gestational diabetes, and type 2 diabetes (T2D) in later life. [42]
Folate is required for the body to make DNA and RNA and metabolise amino acids necessary for cell division and maturation of blood cells. [1] [8] As the human body cannot make folate, it is required in the diet, making it an essential nutrient. [9] It occurs naturally in many foods.
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The methionine gene product MetR and the methionine intermediate homocysteine are known to positively regulate glyA. Homocysteine is a coactivator of glyA and must act in concert with MetR. [15] [16] On the other hand, PurR, a protein which plays a role in purine synthesis and S-adeno-sylmethionine are known to down regulate glyA.
One of the alternate reactions involving CBS is the condensation of cysteine with homocysteine to form cystathionine and hydrogen sulfide (H 2 S). [13] H 2 S in the brain is produced from L-cysteine by CBS. This alternative metabolic pathway is also dependent on adoMet. [14] CBS enzyme activity is not found in all tissues and cells.
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