Search results
Results from the WOW.Com Content Network
Tums (stylized as TUMS) is an antacid made of sucrose (table sugar) and calcium carbonate (CaCO 3) manufactured by Haleon in St. Louis, Missouri, US. They are also available in a sugar-free version. It is an over-the-counter drug, available at many retail stores, including drug stores, grocery stores and mass merchandisers.
The post How Tums Antacid Became America’s Most Trusted Brand appeared first on Reader's Digest. These tiny tablets have been providing relief to overfull tummies for nearly a century, earning ...
Nephrotoxicity is toxicity in the kidneys. It is a poisonous effect of some substances, both toxic chemicals and medications, on kidney function. [1] There are various forms, [2] and some drugs may affect kidney function in more than one way. Nephrotoxins are substances displaying nephrotoxicity.
Kidney dishes made from stainless steel, cardboard and enamelled iron Stainless-steel kidney dish Stack of disposable pulp kidney dishes, stored in a drawer. A kidney dish (British English) or emesis basin (American English) is a shallow basin with a kidney-shaped base and sloping walls used in medical and surgical wards to receive soiled dressings and other medical waste.
Medullary sponge kidney is a congenital disorder of the kidneys characterized by cystic dilatation of the collecting tubules in one or both kidneys. Individuals with medullary sponge kidney are at increased risk for kidney stones and urinary tract infection (UTI). Patients with MSK typically pass twice as many stones per year as do other stone ...
After a kidney disease battle that included weight loss, a new kidney and a whole lot of self-reflection, Atkinson hopes to raise awareness about the kidney disease signs and lifestyle risk ...
Antacids are known to interact with several oral medications, including fluoroquinolone and tetracycline antibiotics, iron, itraconazole, and prednisone. [15] Metal chelation is responsible for some of these interactions (e.g. fluoroquinolones , tetracyclines ), leading to decreased absorption of the chelated drug.
The signs and symptoms of milk-alkali syndrome can develop after only a few days and up to several months following the initial ingestion of absorbable calcium and alkali. [3] However, the severity of signs and symptoms of milk-alkali syndrome is largely dependent upon the duration and quantity of calcium and alkali ingested.