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Isoprenaline was discovered in 1940 [5] and was developed in the 1940s. [7] It was first approved for medical use in 1947 in the United States. [15] Isoprenaline was one of the first synthetic sympathomimetic amines, was the first selective β-adrenergic receptor agonist, and was the first major sympathomimetic agent devoid of pressor effects ...
A stroke is like the brain’s version of a heart attack — and is also life-threatening. When the blood flow to the heart is blocked, we call it a heart attack. And when blood flow to the brain ...
Hemorrhagic stroke is a rare but serious complication of thrombolytic therapy. If a patient has had thrombolysis before, an allergy against the thrombolytic drug may have developed (especially after streptokinase). If the symptoms are mild, the infusion is stopped and the patient is commenced on an antihistamine before infusion is recommenced.
Isoprenaline (non-selective) Norepinephrine reuptake inhibitors , like atomoxetine , methylphenidate , and cocaine , as well as norepinephrine releasing agents , like amphetamine , ephedrine , pseudoephedrine , and phenylpropanolamine , indirectly activate β 1 -adrenergic receptors (as well as other adrenergic receptors) by increasing ...
Aside from pain treatment, doctors may prescribe aspirin to patients who are at risk of heart attack or stroke, according to Harvard Health. These two serious health conditions occur when plaque ...
Stroke is the 5th-leading cause of death in the U.S. and a leading cause of severe disability. On average, a person dies from stroke every 4 minutes. He was given hours to live after stroke. 17 ...
The incidence of post-stroke depression peaks at 3–6 months and usually resolves within 1–2 years after the stroke, although a minority of patients can go on to develop chronic depression. The diagnosis of post-stroke depression is complicated by other consequences of stroke such as fatigue and psychomotor retardation – which do not ...
The ischemic (ischaemic) cascade is a series of biochemical reactions that are initiated in the brain and other aerobic tissues after seconds to minutes of ischemia (inadequate blood supply). [1] This is typically secondary to stroke , injury, or cardiac arrest due to heart attack .