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Examples of such triggers of asthma include naturally occurring aeroallergens like house dust mites, animal feces and pollen. [21] Pets, molds and pests are also potential triggers. [22] When an asthma patient inhales or come into contact withsuch allergen, mast cells in the airway tract releases vasoactive amines and proteases.
However, asthma is rarely the only symptom, and not all people with food or other allergies have asthma [17] Sulfite sensitivity Asthma can occur in reaction to ingestion or inhalation of sulfites, which are added to foods and wine as preservatives. [18] Salicylate sensitivity Salicylates can trigger asthma in sensitive individuals. Salicylates ...
Other potential triggers include medications such as aspirin and beta blockers. [4] Diagnosis is usually based on the pattern of symptoms, response to therapy over time, and spirometry lung function testing. [5] Asthma is classified according to the frequency of symptoms of forced expiratory volume in one second (FEV 1), and peak expiratory ...
The allergy is common [38] [39] and can trigger allergic reactions such as asthma, eczema, or itching. The mite's gut contains potent digestive enzymes (notably peptidase 1) that persist in their feces and are major inducers of allergic reactions such as wheezing. The mite's exoskeleton can also contribute to allergic reactions.
An allergen is an otherwise harmless substance that triggers an allergic reaction in sensitive individuals by stimulating an immune response.. In technical terms, an allergen is an antigen that is capable of stimulating a type-I hypersensitivity reaction in atopic individuals through immunoglobulin E (IgE) responses. [1]
Thunderstorm asthma (also referred to in the media as thunder fever or a pollen bomb [1]) is the triggering of an asthma attack by environmental conditions directly caused by a local thunderstorm. Due to the acute nature of the onset and wide exposure of local populations to the same triggering conditions, severe epidemic thunderstorm asthma ...
Sensitizer-induced occupational asthma is an immunologic form of asthma which occurs due to inhalation of specific substances (i.e., high-molecular-weight proteins from plants and animal origins, or low-molecular-weight agents that include chemicals, metals and wood dusts) and occurs after a latency period of several weeks to years. [1]
Aspirin-exacerbated respiratory disease (AERD), also called NSAID-exacerbated respiratory disease (N-ERD) or historically aspirin-induced asthma and Samter's Triad, is a long-term disease defined by three simultaneous symptoms: asthma, chronic rhinosinusitis with nasal polyps, and intolerance of aspirin and other nonsteroidal anti-inflammatory drugs (NSAIDs).