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Perhaps the most important question now about COVID-19 is the degree to which a prior infection protects from a second infection by the new coronavirus. This affects vaccine development and herd ...
As COVID-19 infections increased among young people, the CDC released updated guidance on Thursday for who is most at risk for severe infection from the coronavirus, noting that there's not a "cut ...
Dr. David Itkin, a Seacoast area infectious disease expert, said having certain people appear to be immune to a particular virus is not new. Can you be 'super-immune' to COVID-19? Unlikely ...
SARS-CoV-2 is the seventh known coronavirus to infect people, after 229E, NL63, OC43, HKU1, MERS-CoV, and the original SARS-CoV. [105] Like the SARS-related coronavirus implicated in the 2003 SARS outbreak, SARS‑CoV‑2 is a member of the subgenus Sarbecovirus (beta-CoV lineage B). [106] [107] Coronaviruses undergo frequent recombination. [108]
Fever in the first week of a COVID-19 infection is part of the body's natural immune response; however in severe cases, if the infections develop into a cytokine storm the fever is counterproductive. As of September 2020, little research had focused on relating fever intensity to outcomes.
A meta-analysis looking into COVID-19 vaccine differences in immunosuppressed individuals found that people with a weakened immune system are less able to produce neutralizing antibodies. For example, organ transplant recipients need three vaccines to achieve seroconversion . [ 409 ]
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Becoming seropositive for COVID-19 antibodies can occur due to either infection with COVID-19 itself or due to becoming vaccinated to COVID-19. [33] Being seropositive for COVID-19 does not intrinsically confer immunity or even resistance. However, higher rates of seroconversion are linked to greater clinical efficacy of vaccines.