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Some plaques occur in the brain as a result of aging, but large numbers of plaques and neurofibrillary tangles are characteristic features of Alzheimer's disease. [5] The plaques are highly variable in shape and size; in tissue sections immunostained for Aβ, they comprise a log-normal size distribution curve, with an average plaque area of 400 ...
Scientists are still not clear as to how these two proteins might cause Alzheimer’s disease. ... “Amyloid-beta is a protein that can become sticky and form plaques between brain cells. These ...
New research is contradicting previously held views that only neurons secret beta-amyloid that forms toxic plaques, a marker of Alzheimer's disease in the brain. The study points to another ...
A main theory behind the cause of Alzheimer’s disease is the build-up of the protein amyloid-beta in the brain. ... only one fifth of those with amyloid plaques develop Alzheimer’s disease.
Amyloid proteins like Aβ can be detected with this technique because of their high content of β-sheet structures. [71] Recently, the formation of Aβ fibrils was resolved in different plaque-types in Alzheimer's disease, indicating that plaques transit different stages in their development. [29]
CAA is associated with brain hemorrhages, particularly microhemorrhages.The accumulation of amyloid beta peptide deposits in the blood vessel walls results in damage of the blood vessels and hindrance of normal blood flow, making blood vessels more prone to bleeding [10] Since CAA can be caused by the same amyloid protein that is associated with Alzheimer's dementia, brain bleeds [11] are more ...
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