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  2. Atomoxetine - Wikipedia

    en.wikipedia.org/wiki/Atomoxetine

    In rats, atomoxetine increased prefrontal cortex catecholamine concentrations without altering dopamine levels in the striatum or nucleus accumbens; in contrast, methylphenidate, a dopamine reuptake inhibitor, was found to increase prefrontal, striatal, and accumbal dopamine levels to the same degree.

  3. Management of attention deficit hyperactivity disorder

    en.wikipedia.org/wiki/Management_of_attention...

    Delayed onset of action. Is slightly-modestly less efficacious than methylphenidate and atomoxetine. [149] Buspirone: Buspar [150] Not available No No 5-HT 1A partial agonist: Low [c] Delayed onset of action. Being a 5-HT 1A receptor partial agonist may afford it the ability to increase dopamine release in the prefrontal cortex. [155] [156 ...

  4. Dopamine reuptake inhibitor - Wikipedia

    en.wikipedia.org/wiki/Dopamine_reuptake_inhibitor

    A dopamine reuptake inhibitor (DRI) is a class of drug which acts as a reuptake inhibitor of the monoamine neurotransmitter dopamine by blocking the action of the dopamine transporter (DAT). Reuptake inhibition is achieved when extracellular dopamine not absorbed by the postsynaptic neuron is blocked from re-entering the presynaptic neuron.

  5. Attention deficit hyperactivity disorder - Wikipedia

    en.wikipedia.org/wiki/Attention_deficit...

    [59]: 13 Atomoxetine alleviates ADHD symptoms through norepinephrine reuptake and by indirectly increasing dopamine in the pre-frontal cortex, [253] sharing 70-80% of the brain regions with stimulants in their produced effects. [252] Atomoxetine has been shown to significantly improve academic performance.

  6. ADHD drugs' impact on brain health, quality of life: What we know

    www.aol.com/adhd-drugs-impact-brain-health...

    More specifically, it looked at the effect of methylphenidate (brand names Ritalin, Concerta), a stimulant, and atomoxetine (Strattera), a non-stimulant, on the brain.

  7. Sympathomimetic drug - Wikipedia

    en.wikipedia.org/wiki/Sympathomimetic_drug

    The mechanisms of sympathomimetic drugs can be direct-acting (direct interaction between drug and receptor), such as α-adrenergic agonists, β-adrenergic agonists, and dopaminergic agonists; or indirect-acting (interaction not between drug and receptor), such as MAOIs, COMT inhibitors, release stimulants, and reuptake inhibitors that increase the levels of endogenous catecholamines.

  8. 6 Everyday Activities That Naturally Release Dopamine ... - AOL

    www.aol.com/news/6-everyday-activities-naturally...

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  9. Selective norepinephrine reuptake inhibitor - Wikipedia

    en.wikipedia.org/wiki/Selective_norepinephrine...

    CYP2D6 inhibitors (e.g., fluoxetine, paroxetine and quinidine) can increase exposure for atomoxetine and it can also increase atomoxetine steady-state plasma concentration. Antihypertensive drugs because it can possibly increase the blood pressure; β2-agonist e.g. albuterol because it may increase the efficacy of albuterol on the ...