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Symptoms: Lattice degeneration itself does not cause symptoms: Diagnostic method: The only way to diagnose the condition is with a dilated fundus examination by an eye care provider. A dilated fundus examination is done by administering dilating eye drops in your eyes to expand the pupil so that the retina can be carefully evaluated.
Family history of retinal detachment [10] Previous retinal detachment in the other eye [8] [9] [10] Lattice degeneration: Thinning of the retina, which increases its susceptibility to breaks or tears. [9] [10] [18] Cystic retinal tuft: A small, raised spot present on the retina from birth that increases the risk for tears and detachment. [9] [10]
Incipient atrophy is demarcated by areas of retinal pigment epithelium (RPE) thinning or depigmentation that precede geographic atrophy in the early stages of AMD. In advanced stages of AMD, atrophy of the RPE (geographic atrophy) and/or development of new blood vessels (neovascularization) result in the death of photoreceptors and central ...
The optic nerve contains axons of nerve cells that emerge from the retina, leave the eye at the optic disc, and go to the visual cortex where input from the eye is processed into vision. There are 1.2 million optic nerve fibers that derive from the retinal ganglion cells of the inner retina. [2] Damage to the optic nerve can have different causes:
In contrast, AIR is an acquired condition where the immune system attacks retinal cells, often leading to more rapid vision deterioration and additional symptoms such as photophobia and color vision loss. While both conditions affect the retina, AIR tends to progress faster and can be associated with a history of cancer or systemic inflammation.
An exposure to doses of 30-35 grays (Gy) or more is usually required to induce clinical symptoms, however, retinopathy may develop after as little as 15 Gy of external-beam radiation. A reported safe dose is 30 Gy (3,000 rad): 10 Gy (1,000 rad) per week in five fractions of 2 Gy (200 rad).
Retinal ganglion cells make up the optic nerve and have a long unmyelinated portion, hence the high energy demand and sensitivity to mitochondrial dysfunction. This is especially the case for smaller axons such as those found in the papillomacular bundle of the retina, which transmit information corresponding to the central visual field. The ...
The diagnosis of toxic or nutritional optic neuropathy is usually established by a detailed medical history and careful eye examination. If the medical history clearly points to a cause, neuroimaging to rule out a compressive or infiltrative lesion is optional. However, if the medical history is atypical or does not clearly point to a cause ...
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