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The Frank-Starling mechanism occurs as the result of the length-tension relationship observed in striated muscle, including for example skeletal muscles, arthropod muscle [4] and cardiac (heart) muscle. [5] [6] [7] As striated muscle is stretched, active tension is created by altering the overlap of thick and thin filaments. The greatest ...
Positive Bowditch effect causes an increase in cardiac output due to the increased force of contraction of heart muscles. [7]This phenomenon is usually absent or even reversed (negative Bowditch effect) in heart failure and other diseases of heart, such as cardiomyopathy and coronary artery disease.
As tension increases in the muscle fibers, the pennation angle also increases. A greater pennation angle results in a smaller force being transmitted to the tendon. [9] Muscle architecture affects the force-velocity relationship. Components of this relationship are fiber length, number of sarcomeres and pennation angle.
Depiction of smooth muscle contraction. Muscle contraction is the activation of tension-generating sites within muscle cells. [1] [2] In physiology, muscle contraction does not necessarily mean muscle shortening because muscle tension can be produced without changes in muscle length, such as when holding something heavy in the same position. [1]
Similarly, the higher the contraction velocity, the lower the tension in the muscle. This hyperbolic form has been found to fit the empirical constant only during isotonic contractions near resting length. [1] The muscle tension decreases as the shortening velocity increases. This feature has been attributed to two main causes.
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Myocardial contractility represents the innate ability of the heart muscle (cardiac muscle or myocardium) to contract.It is the maximum attainable value for the force of contraction of a given heart.
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