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Left ventricular thrombus is a blood clot in the left ventricle of the heart. LVT is a common complication of acute myocardial infarction (AMI). [ 1 ] [ 2 ] Typically the clot is a mural thrombus, meaning it is on the wall of the ventricle. [ 3 ]
Left ventricular mass or LVM refers to the mass of the left ventricle of the heart.. Left ventricular mass index or LVMi is LVM divided by body surface area.. LVM is usually estimated using linear measurements obtained from echocardiography, [1] but can also be calculated using CT or MRI images. [2]
The commonly used method to diagnose LVH is echocardiography, with which the thickness of the muscle of the heart can be measured.The electrocardiogram (ECG) often shows signs of increased voltage from the heart in individuals with LVH, so this is often used as a screening test to determine who should undergo further testing.
Myocardial rupture is most common three to seven days after myocardial infarction, commonly of small degree, but may occur one day to three weeks later. In the modern era of early revascularization and intensive pharmacotherapy as treatment for MI, the incidence of myocardial rupture is about 1% of all MIs. [6]
When the left ventricle is severely impaired before operation (ejection fraction below 30%), however, benefits are less impressive in terms of segmental wall movement but still significant because other parameters might improve as LV function improves; the pulmonary hypertension might be relieved and lengthen survival.
The American Heart Association's 2006 classification of cardiomyopathies considers noncompaction cardiomyopathy a genetic cardiomyopathy. [5] Mutations in LDB3 (also known as "Cypher/ZASP") have been described in patients with the condition. [6] There is recent information in which NCC has been seen in combination with 1q21.1 deletion Syndrome. [7]
Ventricular aneurysms are one of the many complications that may occur after a heart attack.The word aneurysm refers to a bulge or 'pocketing' of the wall or lining of a vessel commonly occurring in the blood vessels at the base of the septum, or within the aorta.
The formation is thought to be primarily due to blood flow stasis. Classically, the thromboembolism lodges at the iliac trifurcation of the aorta, occluding either one or both of the common iliac arteries. Because this split is called the saddle, and is the most frequent location for the thrombus, FATE is commonly known as saddle thrombus. [92]