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Carotidynia is a syndrome marked by soreness of the carotid artery near the bifurcation. Carotid stenosis may occur in patients with atherosclerosis. The intima-media thickness of the carotid artery wall is a marker of subclinical atherosclerosis and it increases with age and with long-term exposure to particulate air pollution. [9]
In human anatomy, the internal and external carotid arise from the common carotid artery, where it bifurcates at cervical vertebrae C3 or C4. The internal carotid artery supplies the brain, including the eyes, [2] while the external carotid nourishes other portions of the head, such as the face, scalp, skull, and meninges.
The carotid body is a small cluster of peripheral chemoreceptor cells and supporting sustentacular cells situated at the bifurcation of each common carotid artery in its tunica externa. [ 1 ] [ 2 ] The carotid body detects changes in the composition of arterial blood flowing through it, mainly the partial pressure of arterial oxygen , but also ...
Carotidynia is a syndrome characterized by unilateral (one-sided) tenderness of the carotid artery, near the bifurcation. It was first described in 1927 by Temple Fay. [1] The most common cause of carotidynia may be migraine, and then it is usually self-correcting. Common migraine treatments may help alleviate the carotidynia symptoms.
The carotid sinus baroreceptor can be oversensitive to manual stimulation from the pressure applied at the carotid sinus at the carotid bifurcation. It is a condition known as 'carotid sinus hypersensitivity' (CSH), 'carotid sinus syndrome' or 'carotid sinus syncope', in which manual stimulation causes large changes in heart rate and blood ...
The internal carotid artery supplies the brain, and the external carotid artery supplies the face. This fork is a common site for atherosclerosis, an inflammatory build-up of atheromatous plaque inside the common carotid artery, or the internal carotid arteries that causes them to narrow. [3] [4]
It runs downward anterior to the internal carotid artery. It communicates with the vagus nerve and sympathetic trunk before dividing in the angle of the bifurcation of the common carotid artery to innervate the carotid body, and carotid sinus.
The fetal carotid origin of the PCA usually regresses as the vertebral and basilar arteries become dominant and it finds a new origin in the basilar artery. About 20% of adults retain PCA origin from the posterior communicating artery, and in turn, the internal carotid arteries. [4]