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Ocular ischemic syndrome is the constellation of ocular signs and symptoms secondary to severe, chronic arterial hypoperfusion to the eye. [1] Amaurosis fugax is a form of acute vision loss caused by reduced blood flow to the eye; it may be a warning sign of an impending stroke, as both stroke and retinal artery occlusion can be caused by thromboembolism due to atherosclerosis elsewhere in the ...
Left untreated, this event carries a high risk of stroke; after carotid endarterectomy, which has a low operative risk, there is a very low postoperative stroke rate." [ 43 ] However, the rate of subsequent stroke after amaurosis is significantly less than after a hemispheric TIA, therefore there remains debate as to the precise indications for ...
[2] [3] Signs and symptoms often appear soon after the stroke has occurred. [3] If symptoms last less than 24 hours, the stroke is a transient ischemic attack (TIA), also called a mini-stroke. [3] Hemorrhagic stroke may also be associated with a severe headache. [3] The symptoms of stroke can be permanent. [5]
Based on symptoms and blood tests, confirmed by biopsy of the temporal artery [4] Differential diagnosis: Takayasu arteritis, [5] stroke, primary amyloidosis [6] Treatment: Steroids, bisphosphonates, proton-pump inhibitor [4] Prognosis: Life expectancy (typically normal) [4] Frequency ~ 1 in 15,000 people a year (> 50 years old) [2]
A transient ischemic attack (TIA), commonly known as a mini-stroke, is a temporary (transient) stroke with noticeable symptoms that end within 24 hours. A TIA causes the same symptoms associated with a stroke, such as weakness or numbness on one side of the body, sudden dimming or loss of vision, difficulty speaking or understanding language or slurred speech.
The MCA is the most common site for the occurrence of ischemic stroke. [1] Depending upon the location and severity of the occlusion, signs and symptoms may vary within the population affected with MCA syndrome. More distal blockages tend to produce milder deficits due to more extensive branching of the artery and less ischemic response.
In children, numerous nonspecific signs and symptoms may be present. [7] The increased pressure leads to compression and traction of the cranial nerves, a group of nerves that arise from the brain stem and supply the face and neck. Most commonly, the abducens nerve (sixth nerve) is involved. This nerve supplies the muscle that pulls the eye ...
Although it is an autosomal recessive disorder, heterozygotes may still manifest much attenuated symptoms. [3] Autosomal dominant inheritance also being reported in a family. [ 4 ] Recently a variant of OPA1 mutation with phenotypic presentation like Behr syndrome is also described. [ 5 ]
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