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Periapical periodontitis of some form is a very common condition. The prevalence of periapical periodontitis is generally reported to vary according to age group, e.g. 33% in those aged 20–30, 40% in 30- to 40-year-olds, 48% in 40- to 50-year-olds, 57% in 50- to 60-year-olds and 62% in those over the age of 60. [ 13 ]
Historically, the cause of nasopalatine duct cysts has been somewhat of an enigma. Although it was originally postulated that the cyst formed from trapped epithelial cells during embryonic fusion of the palatal bones, [3] it is now thought that it forms from oronasal ducts present within the incisive canals.
Periradicular surgery should be very considered where previous endodontic treatment has failed, and possible re-root treatment is the preferred option. [4] If re-root treatment is not possible, will not correct the problem or patient factors prevent it, periradicular surgery is indicated.
Periapical cysts are labeled at left. Periapical cysts comprise approximately 75% of the types of cysts found in the oral region. The ratio of individuals diagnosed with periapical cysts is 3:2 male to female, as well as individuals between 20 and 60 years old. Periapical cysts occur worldwide. Types of Periapical cysts: Apical: 70% Lateral: 20%
Dental radiographs, commonly known as X-rays, are radiographs used to diagnose hidden dental structures, malignant or benign masses, bone loss, and cavities.. A radiographic image is formed by a controlled burst of X-ray radiation which penetrates oral structures at different levels, depending on varying anatomical densities, before striking the film or sensor.
In a study, 14.7% of patients were symptomatic, with pain being the most common symptom and may be associated with osteomyelitis. [2] Other symptoms may include hypoesthesia, paresthesia, and discomfort. [3] In rare instances, patients may experience associated facial swelling with the presence or absence of pain due to concurrent infection. [4]
In 1890, W.D. Miller, considered the father of oral microbiology, was the first to associate pulpal disease with the presence of bacteria. [11] This was confirmed by Kakehashi, who, in 1965, proved that bacteria were the cause of pulpal and periradicular disease in studies using animal models; pulpal exposures were initiated in both normal and germ-free rats, and while no pathologic changes ...
A phoenix abscess is an acute exacerbation of a chronic periapical lesion. It is a dental abscess that can occur immediately following root canal treatment.Another cause is due to untreated necrotic pulp (chronic apical periodontitis). [1]