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  2. Your Gout Guide: From Symptoms to Treatment - AOL

    www.aol.com/gout-guide-symptoms-treatment...

    Gout in foot joints is most common, with gout often affecting the big toe joint. However, other joints can be affected as well, particularly those in the lower body. For example, gout in ankles or ...

  3. Diclofenac - Wikipedia

    en.wikipedia.org/wiki/Diclofenac

    Diclofenac, sold under the brand name Voltaren among others, is a nonsteroidal anti-inflammatory drug (NSAID) used to treat pain and inflammatory diseases such as gout. [6] [9] It can be taken orally (swallowed by mouth), inserted rectally as a suppository, injected intramuscularly, injected intravenously, applied to the skin topically, or through eye drops.

  4. Losartan - Wikipedia

    en.wikipedia.org/wiki/Losartan

    It comes as a tablet that is taken by mouth. [4] It may be used alone or in addition to other blood pressure medication. [4] Up to six weeks may be required for the full effects to occur. [4] Common adverse effects include muscle cramps, stuffy nose, dizziness, cough, high blood potassium, and anemia. [4]

  5. Furosemide - Wikipedia

    en.wikipedia.org/wiki/Furosemide

    Furosemide also can lead to gout caused by hyperuricemia. Hyperglycemia is also a common side effect. [28] [29] [30] The tendency, as for all loop diuretics, to cause low serum potassium concentration (hypokalemia) has given rise to combination products, either with potassium or with the potassium-sparing diuretic amiloride (Co-amilofruse ...

  6. Potassium chloride (medical use) - Wikipedia

    en.wikipedia.org/wiki/Potassium_chloride...

    Potassium chloride, also known as potassium salt, is used as a medication to treat and prevent low blood potassium. [2] Low blood potassium may occur due to vomiting, diarrhea, or certain medications. [3] The concentrated version should be diluted before use. [2] It is given by slow injection into a vein or by mouth. [4]

  7. Patiromer - Wikipedia

    en.wikipedia.org/wiki/Patiromer

    Patiromer works by binding free potassium ions in the gastrointestinal tract and releasing calcium ions for exchange, thus lowering the amount of potassium available for absorption into the bloodstream and increasing the amount that is excreted via the feces. The net effect is a reduction of potassium levels in the blood serum. [4] [11]

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