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Reduced affect display, sometimes referred to as emotional blunting or emotional numbing, is a condition of reduced emotional reactivity in an individual. It manifests as a failure to express feelings either verbally or nonverbally, especially when talking about issues that would normally be expected to engage emotions.
Schizophrenia in general causes abnormalities in emotional understanding of individuals, all of which are clinically considered as an emotional blunting symptom. Individuals with schizophrenia show less emotional experiences, display less emotional expressions, and fail to recognize the emotional experiences and/or expressions of other individuals.
Disorders involving these reduced affect displays most commonly include schizophrenia, post traumatic stress disorder, depression, autism and persons with traumatic brain injuries. [20] One study has shown that people with schizophrenia that experience flat affect, can also experience difficulty perceiving the emotions of a healthy individual.
Research has shown that failures in emotional regulation may be related to the display of acting out, externalizing disorders, or behavior problems. When presented with challenging tasks, children who were found to have defects in emotional regulation (high-risk) spent less time attending to tasks and more time throwing tantrums or fretting ...
Affect display is a critical facet of interpersonal communication. Evolutionary psychologists have advanced the hypothesis that hominids have evolved with sophisticated capability of reading affect displays. [45] Emotions are portrayed as dynamic processes that mediate the individual's relation to a continually changing social environment. [46]
Simple-type schizophrenia is characterized by negative ("deficit") symptoms, such as avolition, apathy, anhedonia, reduced affect display, lack of initiative, lack of motivation, low activity; with absence of hallucinations or delusions of any kind.
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However, damage to or reduced functioning of the anterior cingulate cortex (ACC) and striatum have been especially implicated in DDM. [3] [8] [4] The striatum is part of the dopaminergic mesolimbic pathway, which connects the ventral tegmental area (VTA) of the midbrain to the nucleus accumbens (NAc) of the ventral striatum and basal ganglia.