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Electrical waves track a systole (a contraction) of the heart. The end-point of the P wave depolarization is the start-point of the atrial stage of systole. The ventricular stage of systole begins at the R peak of the QRS wave complex; the T wave indicates the end of ventricular contraction, after which ventricular relaxation (ventricular diastole) begins.
Ventricular systole is the contractions, following electrical stimulations, of the ventricular syncytium of cardiac muscle cells in the left and right ventricles. Contractions in the right ventricle provide pulmonary circulation by pulsing oxygen-depleted blood through the pulmonary valve then through the pulmonary arteries to the lungs.
The faster the contractions are, the shorter the filling time and both the EDV and preload are lower. [1] The relationship between ventricular stretch and contraction has been stated in the Frank-Starling mechanism which says that the force of contraction is directly proportional to the initial length of muscle fibre. So that the greater the ...
The left ventricular muscle must relax and contract quickly and be able to increase or lower its pumping capacity under the control of the nervous system. In the diastolic phase, it has to relax very quickly after each contraction so as to quickly fill with the oxygenated blood flowing from the pulmonary veins. Likewise in the systolic phase ...
During the ventricular contraction portion of the cardiac cycle, the Purkinje fibers carry the contraction impulse from both the left and right bundle branch to the myocardium of the ventricles. [5] This causes the muscle tissue of the ventricles to contract.
A ventricular septal defect is a hole between the two lower chambers of the heart Tricuspid atresia. The tricuspid valve is located between the right atrium and the right ventricle.
Type A receptors are activated by wall tension, which occurs via atrial contraction during ventricular diastole. Type B receptors are activated by wall stretch, which occurs via atrial filling during ventricular systole. [1] In the right atrium, the stretch receptors occur at the junction of the venae cavae.
As a result, the heart has a hard time pumping blood through the lungs, and the blood vessels eventually undergoes fibrosis. The increased workload on the heart causes hypertrophy of the right ventricle, which leads less blood being pump through the lungs and decreased blood to the left side of the heart. As a result of all of this, the left ...