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Animals such as birds and humans have different docking stations on their cells — known as sialic acid receptors. In order for a flu virus to open a door into a cell and begin replicating, it ...
By analogy, the term is used in human reproduction, but more commonly refers to the genetic disorders and other consequences that may arise from expression of deleterious recessive traits resulting from incestuous sexual relationships and consanguinity. Animals avoid inbreeding only rarely. [2]
That’s worrisome, experts say, because bird flu viruses like H5N1 typically attach to cell receptors found in birds and other animals, but not commonly in humans. This is why bird flu typically ...
“In nature, the occurrence of this single mutation could be an indicator of human pandemic risk,” according to an editorial note attached to the paper. The study showed that just one mutation ...
Human and mouse somatic cells have a mutation rate more than ten times higher than the germline mutation rate for both species; mice have a higher rate of both somatic and germline mutations per cell division than humans. The disparity in mutation rate between the germline and somatic tissues likely reflects the greater importance of genome ...
Evolutionary biologists have used mutation accumulation experiments, in which mutations are allowed to drift to fixation in inbred lines, to study the effect of spontaneous mutations on phenotype character. Phenotypic assays significantly determine whether and how quickly population with accumulated deleterious mutational loads can result in ...
The first severe case of bird flu in the U.S. is showing signs of mutation, stoking fears that the virus could become more transmissible among humans, the U.S. Centers for Disease Control and ...
The international pictogram for chemicals that are sensitising, mutagenic, carcinogenic or toxic to reproduction. In genetics, a mutagen is a physical or chemical agent that permanently changes genetic material, usually DNA, in an organism and thus increases the frequency of mutations above the natural background level.