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  2. Mercedes-Benz M272 engine - Wikipedia

    en.wikipedia.org/wiki/Mercedes-Benz_M272_engine

    The Mercedes-Benz M272 engine is an automobile piston V6 engine family used in the 2000s (decade). Introduced in 2004, it is based on the M112 V6 introduced in 1998.. All M272 engines have aluminum engine blocks with a 90° V-angle with silicon/aluminum lined cylinders.

  3. Buick V6 engine - Wikipedia

    en.wikipedia.org/wiki/Buick_V6_engine

    The engine was originally 198 cu in (3.2 L) and was marketed as the Fireball engine. GM continued to develop and refine the 231 cu in (3.8 L) V6, eventually and commonly referred to simply as the 3800, through numerous iterations. The 3800 made the Ward's 10 Best Engines of the 20th Century list and made Ward's yearly 10 Best list numerous ...

  4. Street Fighter III: 3rd Strike - Wikipedia

    en.wikipedia.org/wiki/Street_Fighter_III:_3rd_Strike

    Street Fighter III: 3rd Strike - Fight for the Future [a] is a 1999 fighting game developed and published by Capcom for arcades and ported to the Dreamcast in 2000. In 2004 it was released on the PlayStation 2 and Xbox.

  5. BMW 2 Series - Wikipedia

    en.wikipedia.org/wiki/BMW_2_Series

    The second-generation 2 Series coupé was revealed in July 2021 as the successor to the F22 2 Series coupé and convertible. Remaining a rear-wheel-drive-based model, the G42 is built on the CLAR platform with a roughly 50 50 weight distribution and shares many mechanical components and engine options with the G20 3 Series and G22 4 Series.

  6. The Twilight Saga: Breaking Dawn – Part 2 - Wikipedia

    en.wikipedia.org/wiki/The_Twilight_Saga:_Breaking...

    The Twilight Saga: Breaking Dawn – Part 2 (or simply Breaking Dawn – Part 2) is a 2012 American romantic fantasy film directed by Bill Condon from a screenplay by Melissa Rosenberg, based on the 2008 novel Breaking Dawn by Stephenie Meyer.

  7. Fibroblast growth factor receptor 3 - Wikipedia

    en.wikipedia.org/wiki/Fibroblast_growth_factor...

    Simplification on the mutation 46 XX 4q16.3 (female), 46XY 4q16.3 (male). Gain of function mutations in this gene can develop dysfunctional proteins "impede cartilage growth and development and affect chondrocyte proliferation and calcification" [6] which can lead to craniosynostosis and multiple types of skeletal dysplasia (osteochondrodysplasia).

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