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Gingivitis is a non-destructive disease that causes inflammation of the gums; [1] ulitis is an alternative term. [2] The most common form of gingivitis, and the most common form of periodontal disease overall, is in response to bacterial biofilms (also called plaque) that are attached to tooth surfaces, termed plaque-induced gingivitis.
Bacterial plaque is one of the major causes for dental decay and gum disease. [1] Interestingly, it has been observed that differences in the composition of dental plaque microbiota exist between men and women , particularly in the presence of periodontitis .
Periodontal disease, also known as gum disease, is a set of inflammatory conditions affecting the tissues surrounding the teeth. [5] In its early stage, called gingivitis, the gums become swollen and red and may bleed. [5] It is considered the main cause of tooth loss for adults worldwide.
What are the common causes of gum disease? Gum disease is typically caused by an accumulation of bacterial plaque — a sticky, colorless film — on the teeth and gums due to poor oral hygiene ...
Gingival enlargement has a multitude of causes. The most common is chronic inflammatory gingival enlargement, when the gingivae are soft and discolored. This is caused by tissue edema and infective cellular infiltration caused by prolonged exposure to bacterial plaque, and is treated with conventional periodontal treatment, such as scaling and root planing.
Gum disease is typically caused by a bacterial infection that results from an overgrowth of dental plaque — a sticky, colorless film — on the teeth and gums.
Dental plaque accumulates at the surfaces when proper cleaning and maintaining is not done. There is inflammation due to the bacteria released from the toxins. calculus forms and if not removed, causes this disease. [5] In most cases, the cause of gingival diseases such as gingivitis are due to poor oral hygiene.
Chronic periodontitis is initiated by Gram-negative tooth-associated microbial biofilms that elicit a host response, which results in bone and soft tissue destruction. In response to endotoxin derived from periodontal pathogens, several osteoclast-related mediators target the destruction of alveolar bone and supporting connective tissue such as the periodontal ligament.
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