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A mild case of trench foot. Nonfreezing cold injury commonly affects the feet due to prolonged exposure to wet socks or cold standing water. [4] Symptoms progress through a series of four stages. [4] [15] A severe case of trench foot. During cold exposure. Affected skin becomes numb, which can cause a clumsy walking pattern if the feet are affected
Fluid builup in the lower legs, feet and hands—also known as pedal edema— occurs because the heart’s ability to pump blood is too weak, which causes the blood to settle and accumulate in tissue.
Paresthesias are usually painless and can occur anywhere on the body, but most commonly occur in the arms and legs. [1] The most familiar kind of paresthesia is the sensation known as "pins and needles" after having a limb "fall asleep". A less well-known and uncommon paresthesia is formication, the sensation of insects crawling on the skin.
Trench foot or immersion foot: a condition caused by repetitive exposure to water at non-freezing temperatures [11] The normal human body temperature is often stated as 36.5–37.5 °C (97.7–99.5 °F). [13] Hyperthermia and fevers are defined as a temperature of greater than 37.5–38.3 °C (99.5–100.9 °F). [8]
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Areas that are usually affected include cheeks, ears, nose and fingers and toes. Frostbite is often preceded by frostnip. [2] The symptoms of frostbite progress with prolonged exposure to cold. Historically, frostbite has been classified by degrees according to skin and sensation changes, similar to burn classifications.
The lateral femoral cutaneous nerve (LFCN) is a purely sensory nerve, [3] [2] and consequently the symptoms are also sensory. [4] Symptoms are typically unilateral, seen in about 78% of cases, but may be bilateral. [4] [2] The most common symptom is pain, paresthesias, or dysthesias on the anterolateral surface of the thigh that extends just ...
The combination of increased pain signaling and degeneration of pain-transmitting fibers leads to a variable condition with signs and symptoms that can change over time. SCN9A gene mutations have been found in approximately 30 percent of individuals with small fiber neuropathy; SCN10A gene mutations are responsible for about 5 percent of cases.