Search results
Results from the WOW.Com Content Network
Anterograde amnesia can also be caused by alcohol intoxication, a phenomenon commonly known as a blackout. Studies show rapid rises in blood alcohol concentration over a short period of time severely impair or in some cases completely block the brain's ability to transfer short-term memories created during the period of intoxication to long ...
Kent Cochrane was born on August 5, 1951, as the oldest of five children. They grew up in the suburbs of Toronto, Ontario.After attending a community college to study business administration, he obtained a quality control job at a manufacturing plant, which he held until the time of his motorcycle accident.
Head trauma is a very broad range as it deals with any kind of injury or active action toward the brain which might cause amnesia. Retrograde and anterograde amnesia is more often seen from events like this, an exact example of a cause of the two would be electroconvulsive therapy, which would cause both briefly for the receiving patient.
Although a TGA is scary for both patients and their families, transient global amnesia is a benign condition with an excellent prognosis. Jeff Hersh, Ph.D., M.D., can be reached at DrHersh@juno.com .
Anterograde amnesia is one type of memory loss where people have difficulty forming new memories after the amnesia-causing event. Anterograde amnesia is one type of memory loss where people have ...
Patient N.A. (born July 9th, 1938) was an American man who developed anterograde amnesia as a result of a fencing accident. He was a patient studied by Larry Squire, a professor of psychiatry, neuroscience and psychology at the University of California. The cause of his amnesia was found to be a thalamic lesion extending to the hypothalamus.
Amnesia is an abnormal mental state in which memory and learning are affected out of all proportion to other cognitive functions in an otherwise alert and responsive patient. [5] There are two forms of amnesia: Anterograde amnesia and retrograde amnesia, that show hippocampal or medial temporal lobe damage.
Henry Gustav Molaison (February 26, 1926 – December 2, 2008), known widely as H.M., was an American who had a bilateral medial temporal lobectomy to surgically resect the anterior two thirds of his hippocampi, parahippocampal cortices, entorhinal cortices, piriform cortices, and amygdalae in an attempt to cure his epilepsy.