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One of the important questions in sleep research is clearly defining the sleep state. This problem arises because sleep was traditionally defined as a state of consciousness and not as a physiological state, [14] [15] thus there was no clear definition of what minimum set of events constitute sleep and distinguish it from other states of partial or no consciousness.
The use of neurotransmitters to increase potentiation of synaptic pathways specifically at CA1 and CA3 pyramidal cells is a promising way to treat nightmares. Sleep deprivation increases the expression of HDAC2 which in turn leads to a significant deficiency in histone acetylation. [4]
[13] [14] The VLPO is inhibited during wakefulness by the arousal-inducing neurotransmitters norepinephrine and acetylcholine. [9] [15] The role of the VLPO in sleep and wakefulness, and its association with sleep disorders – particularly insomnia and narcolepsy – is a growing area of neuroscience research.
Improvement to cognitive performance caused by exercise could last for 24 hours, a new study shows. Scientists also linked getting 6 or more hours of sleep to better memory test scores the next day.
A neurotransmitter is a signaling molecule secreted by a neuron to affect ... sleep, memory and ... Drugs can influence behavior by altering neurotransmitter activity
The areas activated during REM sleep are approximately inverse to those activated during non-REM sleep [16] and display greater activity than in quiet waking. The "anterior paralimbic REM activation area" (APRA) includes areas linked with emotion , memory, fear and sex, and may thus relate to the experience of dreaming during REMS.
Scientists observed that slow synchronized oscillations of the neurotransmitter norepinephrine, along with cerebral blood and cerebrospinal fluid (CSF), combine during non-rapid eye movement sleep ...
These disturbances cause the normal physiology of sleep to change, such as the biological clock and body temperature during rest. [44] Patients with AD experience insomnia, hypersomnia, and other sleep disorders as a result of the degeneration of the SCN and changes in critical neurotransmitter concentrations. [44]