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Chest pain due to coronary ischemia commonly radiates to the arm or neck. [7] Certain individuals such as women, diabetics, and the elderly may present with more varied symptoms. [ 8 ] If blood flow through the coronary arteries is stopped completely, cardiac muscle cells may die, known as a myocardial infarction, or heart attack.
By far the most dramatic is rupture of the free wall of the left or right ventricles, as this is associated with immediate hemodynamic collapse and death secondary to acute pericardial tamponade. Rupture of the interventricular septum will cause a ventricular septal defect. Rupture of a papillary muscle will cause acute mitral regurgitation.
A popular theory termed the "no flow, no grow" hypothesis suggest that primary anatomic defects of the aortic and mitral valves lead to malformations of the left ventricle and its outflow tract. [9] These primary defects can be divided into those that lead to outflow tract obstruction or reduced left ventricular filling. [ 7 ]
In experimental animal models in pigs studying impacts by a hard ball to the chest wall, impacts that occurred directly over the center of the left ventricle, where there is no overlying lung tissue, were the most likely to cause ventricular fibrillation. [1] Impacts not over the heart did not cause ventricular fibrillation. [1]
However, in acute necrotizing eosinophilic myocarditis, echocardiography usually gives diagnostically helpful evidence of a non-enlarged heart with a thickened and poorly contracting left ventricle. Gadolinium -based cardiac magnetic resonance imaging is the most useful non-invasive procedure for diagnosing eosinophilic myocarditis.
Inferior left ventricle wall scar, short axis echocardiography view Myocardial scarring is the accumulation of fibrous tissue resulting after some form of trauma to the cardiac tissue. [ 1 ] [ 2 ] Fibrosis is the formation of excess tissue in replacement of necrotic or extensively damaged tissue.
EKG depiction of left ventricular hypertrophy. Left ventricular hypertrophy is a leading cause of sudden cardiac deaths in the adult population. [39] [30] This is most commonly the result of longstanding high blood pressure, or hypertension, which has led to maladaptive overgrowth of muscular tissue of the left ventricle, the heart's main ...
Rhythmicity and contractility of the heart may be normal, but the stiff walls of the heart chambers (atria and ventricles) keep them from adequately filling, reducing preload and end-diastolic volume. Thus, blood flow is reduced, and blood volume that would normally enter the heart is backed up in the circulatory system.