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A popular theory termed the "no flow, no grow" hypothesis suggest that primary anatomic defects of the aortic and mitral valves lead to malformations of the left ventricle and its outflow tract. [9] These primary defects can be divided into those that lead to outflow tract obstruction or reduced left ventricular filling. [ 7 ]
During the course of evaluation of the patient, a bulging out of the left ventricular apex with a hypercontractile base of the left ventricle is often noted. It is the hallmark bulging-out of the apex of the heart with preserved function of the base that earned the syndrome the name takotsubo ("octopus trap") in Japan, where it was first described.
This process is particularly apparent in the ventricles, and particularly so in the left ventricle. Noncompaction cardiomyopathy results when there is failure of this process of compaction. Because the consequence of non-compaction is particularly evident in the left ventricle, the condition is also called left ventricular noncompaction. Other ...
Broken heart syndrome is caused by extreme emotional or physical stress. [3] Treatment depends on the type of cardiomyopathy and the severity of symptoms. [5] Treatments may include lifestyle changes, medications, or surgery. [5] Surgery may include a ventricular assist device or heart transplant. [5]
The left ventricle is the most common location of cardiac fibroma and accounts for approximately 57% of cardiac fibroma cases followed by the right ventricle with 27.5% of cases. [1] Symptoms of the disease depend on the size of the tumor, its location relative to the conduction system, and whether it obstructs blood flow.
The pain associated with microvascular angina is normally more intense and it lasts for longer periods of time compared to pain caused by other conditions. Many gastric conditions can cause chest pains (sub-sternal pain), while this is usually associated with consumption of food this is not always the case, and is a very common differential ...
Late gadolinium enhancement shows an increased signal of the mid wall at the inferolateral wall of the base of the left ventricle, usually in the non-hypertrophic ventricle. T1-weighted imaging can show low T1 signal due to sphingolipid storage in the heart even without ventricular hypertrophy in 40% of those affected by the disease.
Shone's syndrome is a rare congenital heart defect described by Shone in 1963. In the complete form, four left-sided defects are present: [1] Supravalvular mitral membrane (SVMM) Parachute mitral valve; Subaortic stenosis (membranous or muscular) Coarctation of the aorta