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These include issues around the governance of the therapy, whether treatment should be available only to those who can afford it, and whether the availability of treatment creates a stigma for those with color blindness. Given the large number of people with color blindness, there is also the question of whether color blindness is a disorder. [16]
Blue cone monochromacy (BCM) is an inherited eye disease that causes severe color blindness, poor visual acuity, nystagmus, hemeralopia, and photophobia due to the absence of functional red (L) and green (M) cone photoreceptor cells in the retina. BCM is a recessive X-linked disease and almost exclusively affects XY karyotypes.
Males are more likely to inherit red–green color blindness than females, because the genes for the relevant opsins are on the X chromosome. [1] Screening for congenital red–green color blindness is typically performed with the Ishihara or similar color vision test. [1] It is a lifelong condition, and has no known cure or treatment. [1]
Color blindness may also present itself as a symptom of degenerative diseases of the eye, such as cataract and age-related macular degeneration, and as part of the retinal damage caused by diabetes. Vitamin A deficiency may also cause color blindness. [47] Color blindness may be a side effect of prescription drug use.
Far-sightedness primarily affects young children, with rates of 8% at 6 years old and 1% at 15 years old. [9] It then becomes more common again after the age of 40, known as presbyopia, affecting about half of people. [4] The best treatment option to correct hypermetropia due to aphakia is IOL implantation. [2]
Childhood blindness is most prevalent among children with genetic ancestry from Africa and Asia, who represent 75% of the world's affected population. [13] [32] A 2014 review indicated that an estimated 238,500 children with bilateral blindness (rate 1.2/1,000) live in the Eastern Mediterranean region. [30]
The conditions include: monochromatic color blindness, poor visual acuity, and day-blindness. The syndrome is also present in an incomplete form that exhibits milder symptoms, including residual color vision. Achromatopsia is estimated to affect 1 in 30,000 live births worldwide.
The WHO estimated in 1995 that 13.8 million children had some degree of visual loss related to VAD. [10] Night blindness and its worsened condition, xerophthalmia, are markers of Vitamin A deficiency; collections of keratin in the conjunctiva, known as Bitot's spots, and ulceration and necrosis of cornea keratomalacia can be seen.