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Common side effects include nausea, abdominal pain, and constipation. [2] It may worsen the symptoms of asthma. [2] Propranolol may cause harmful effects for the baby if taken during pregnancy; [7] however, its use during breastfeeding is generally considered to be safe. [8] It is a non-selective beta blocker which works by blocking β ...
In the first year of treatment, the rate of growth may increase from half as fast as other children are growing to twice as fast (e.g., from 1 inch a year to 4 inches, or 2.5 cm to 10). Growth typically slows in subsequent years, but usually remains above normal so that over several years a child who had fallen far behind in their height may ...
Nicergoline is known to enhance the cardiac depressive effects of propranolol. [6] At high dosages, it is advisable to seek one's physician's guidance if combining with potent vasodilators such as bromocriptine , Ginkgo biloba , picamilon , vinpocetine or xantinol nicotinate .
Therefore, blocking β 2-adrenoceptors lowers plasma glucose. β 1-blockers have fewer metabolic side effects in diabetic patients; however, the fast heart rate that serves as a warning sign for insulin-induced low blood sugar may be masked, resulting in hypoglycemia unawareness. This is termed beta blocker-induced hypoglycemia unawareness ...
Since this response, which is mostly seen as an increase in blood pressure, is produced by the release of the endogenous adrenergic ligands, administration of an adrenergic antagonist results a decrease in blood pressure, which is controlled by both heart rate and vasculature tone. [14]
In addition to increasing height in children and adolescents, growth hormone has many other effects on the body: Increases calcium retention, [41] [citation needed] and strengthens and increases the mineralization of bone; Increases muscle mass through sarcomere hypertrophy; Promotes lipolysis; Increases protein synthesis
Figure 1: The chemical structure of dichloroisoprenaline or dichloroisoproterenol (), abbreviated DCI — the first β-blocker to be developed. β adrenergic receptor antagonists (also called beta-blockers or β-blockers) were initially developed in the 1960s, for the treatment of angina pectoris but are now also used for hypertension, congestive heart failure and certain arrhythmias. [1]
Medications are used to reverse the symptoms of extrapyramidal side effects caused by antipsychotics or other drugs, by either directly or indirectly increasing dopaminergic neurotransmission. The treatment varies by the type of the EPS, but may involve anticholinergic agents such as procyclidine , benztropine , diphenhydramine , and ...
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