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Cardiogenic pulmonary edema is the result of cardiovascular insufficiency. Treatment is directed at improving cardiovascular function and providing supportive care. [43] [44] Positioning upright may relieve symptoms. A loop diuretic such as furosemide is administered, often together with morphine to reduce respiratory distress. [44]
NPPE develops as a result of significant negative pressure generated in the chest cavity by inspiration against an upper airway obstruction. These negative pressures in the chest lead to increase venous supply to the right side of the heart while simultaneously creating more resistance for the left side of the heart to supply blood to the rest of the body (). [4]
Diffusing capacity of the lung (D L) (also known as transfer factor) measures the transfer of gas from air in the lung, to the red blood cells in lung blood vessels. It is part of a comprehensive series of pulmonary function tests to determine the overall ability of the lung to transport gas into and out of the blood.
When a person is recumbent, or is lying down, blood is redistributed from the lower extremities and abdominal cavity (splanchnic circulation) to the lungs. [5] Failure to accommodate this redistribution results in decreased vital capacity and pulmonary compliance, further causing the shortness of breath experienced in PND. In addition to the ...
Flash Pulmonary Edema or Crash Pulmonary Edema is a clinical characterization of acute heart failure with a dramatic presentation. [4] It is an acute cardiac disease precipitated by cardiac events and usually associated with severe hypertension.
Death from pulmonary edema as the result of circulatory overload following transfusion was reported as early as 1936. [20] However, the term 'transfusion associated circulatory overload' was not coined until the 1990s when it was seen as a separate complication following blood transfusion.
In pulmonary consolidations and infiltrates, air bronchograms are most commonly caused by pneumonia or pulmonary edema (especially with alveolar edema). [2] [3] Other potential causes of consolidations or infiltrates with air bronchograms are: [2] Pulmonary edema; Non-obstructive atelectasis; Severe interstitial lung disease; Pulmonary infarct
These abnormal phenomena are usually seen in chronic bronchitis, asthma, hepatopulmonary syndrome, and acute pulmonary edema. A high V/Q ratio decreases pCO 2 and increases pO 2 in alveoli. Because of the increased dead space ventilation, the arterial pO 2 is reduced and thus also the peripheral oxygen saturation is lower than normal, leading ...
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