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Prior to the findings of the hyperplasia of neuroendocrine cells it was known as tachypnea of infancy, as most children outgrow the need for oxygen supplementation within two to seven years. It is characterized by tachypnea, hypoxemia, and retractions. [1] It is typically diagnosed in infants and children younger than one year of age. [2]
Late onset congenital adrenal hyperplasia (LOCAH), also known as nonclassic congenital adrenal hyperplasia (NCCAH or NCAH), is a milder form of congenital adrenal hyperplasia (CAH), [1] a group of autosomal recessive disorders characterized by impaired cortisol synthesis that leads to variable degrees of postnatal androgen excess.
Congenital adrenal hyperplasia (CAH) is a group of autosomal recessive disorders characterized by impaired cortisol synthesis. [ 1 ] [ 2 ] It results from the deficiency of one of the five enzymes required for the synthesis of cortisol in the adrenal cortex . [ 3 ]
Lipoid congenital adrenal hyperplasia is an endocrine disorder that is an uncommon and potentially lethal form of congenital adrenal hyperplasia (CAH). It arises from defects in the earliest stages of steroid hormone synthesis: the transport of cholesterol into the mitochondria and the conversion of cholesterol to pregnenolone—the first step in the synthesis of all steroid hormones.
Follicular hyperplasia is one of the most common types of benign lymphadenopathies. [1] It can be typically found in children and young adults however all ages are subject to follicular hyperplasia, including the elderly. Lymphadenopathies such as follicular hyperplasia, are usually localized but can also be generalized and are non gender specific.
The U.S. Preventive Services Task Force is recommending for the first time that kids ages 8 and up be screened for anxiety disorder. Yahoo News spoke with a member of the task force to learn more ...
Hyperplasia of the breast – "Hyperplastic" lesions of the breast include usual ductal hyperplasia, a focal expansion of the number of cells in a terminal breast duct, and atypical ductal hyperplasia, in which a more abnormal pattern of growth is seen, and which is associated with an increased risk of developing breast cancer.
Children with CAH often experience increased height during early childhood, but their final adult height tends to be shorter than expected. Advanced bone age and early fusion of growth plates due to excess androgens contribute to this outcome. Additionally, glucocorticoid treatment for CAH can affect growth and result in decreased final height.