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Elevated or normal serum vitamin B 12 levels may also be associated with a functional vitamin deficiency. [1] Functional deficiency has been described despite high B 12 concentrations and is due to a failure of cellular uptake, intracellular processing, trafficking, or utilization. [1] However, low vitamin B 12 levels may occur other than the ...
Evidence exists linking elevated homocysteine levels with vascular dementia [10] and Alzheimer's disease. [11] [12] [13] There is also evidence that elevated homocysteine levels and low levels of vitamin B6 and B12 are risk factors for mild cognitive impairment and dementia. [14]
How fast B 12 levels change depends on the balance between how much B 12 is obtained from the diet, how much is secreted and how much is absorbed. The total amount of vitamin B 12 stored in the body is about 2–5 mg in adults. Around 50% of this is stored in the liver.
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Several serious, even life-threatening diseases cause elevated serum HC, measured as abnormally high serum vitamin B 12. [ 2 ] Transcobalamin II (TC-2) , a nonglycoprotein secretory protein of molecular mass 43 kDa, is encoded in the human by the TCN2 gene.
In the body, homocysteine can be recycled into methionine or converted into cysteine with the aid of vitamin B 6, B 9, and B 12. [3] High levels of homocysteine in the blood (hyperhomocysteinemia) is regarded as a marker of cardiovascular disease, likely working through atherogenesis, which can result in ischemic injury.
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In common forms of MTHFR deficiency, elevated plasma homocysteine levels have sometimes been treated with Vitamin B12 and low doses of folic acid. [2] Although this treatment significantly decreases the serum levels of homocysteine, this treatment is not thought to improve health outcomes. [11] [12] [13]
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