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Dental pulpal testing is a clinical and diagnostic aid used in dentistry to help establish the health of the dental pulp within the pulp chamber and root canals of a tooth. . Such investigations are important in aiding dentists in devising a treatment plan for the tooth being test
The health of the dental pulp can be established by a variety of diagnostic aids which test either the blood supply to a tooth (Vitality Test) or the sensory response of the nerves within the root canal to specific stimuli (Sensitivity Test). Although less accurate, sensitivity tests, such as Electric Pulp Tests or Thermal Tests, are more ...
Test cavity: The preparation of a test cavity involves cutting into the dentine of a tooth to determine whether the sensory element of the pulp is still functioning. Test cavity preparation is a last resort as this method is considered invasive and irreversible.
Pulp necrosis is a clinical diagnostic category indicating the death of cells and tissues in the pulp chamber of a tooth with or without bacterial invasion. [1] It is often the result of many cases of dental trauma , caries and irreversible pulpitis .
Exposure of the pulp causes pulpitis (an inflammation which can become irreversible, leading to pain and pulp necrosis, and necessitating either root canal treatment or extraction). [1] The ultimate goal of pulp capping or stepwise caries removal is to protect a healthy (or reversibly inflammed) dental pulp, and avoid the need for root canal ...
Periodontal abscesses are less common than apical abscesses, but are still frequent. The key difference between the two is that the pulp of the tooth tends to be alive, and will respond normally to pulp tests. However, an untreated periodontal abscess may still cause the pulp to die if it reaches the tooth apex in a periodontic-endodontic lesion.
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In 1890, W.D. Miller, considered the father of oral microbiology, was the first to associate pulpal disease with the presence of bacteria. [11] This was confirmed by Kakehashi, who, in 1965, proved that bacteria were the cause of pulpal and periradicular disease in studies using animal models; pulpal exposures were initiated in both normal and germ-free rats, and while no pathologic changes ...
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