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The adductor magnus is a large triangular muscle, situated on the medial side of the thigh.. It consists of two parts. The portion which arises from the ischiopubic ramus (a small part of the inferior ramus of the pubis, and the inferior ramus of the ischium) is called the pubofemoral portion, adductor portion, or adductor minimus, and the portion arising from the tuberosity of the ischium is ...
A positive Trendelenburg's sign is caused by weakness or ineffective action of the abductor muscles of the lower limb, the gluteus medius muscle and the gluteus minimus muscle. [2] Damage to the motor nerve supply of the lateral gluteal muscles (gluteus medius muscle and gluteus minimus muscle)
Feeling this would indicate an organic cause of the paresis. If the examiner does not feel the "normal" leg's heel pushing down as the patient flexes the hip of the "weak" limb, then this suggests functional weakness (sometimes called "conversion disorder"), i.e. that effort is not being transmitted to either leg. [citation needed]
The adductor magnus tendon is an excellent, consistent landmark because it is rarely injured. The vastus medialis obliquus muscle courses over the anteromedial thigh, attaching along the adductor magnus anterior border and to the quadratus femoris tendon.
The adductor reflex is elicited by tapping over either the medial epicondyle of the femur or the medial condyle of the tibia, which should cause the adductor muscles of the hip to contract, moving the leg inwards. [2] The sign was described by John G Hannington-Kiff in 1980. [3]
The predominant symptoms of NC involve one or both legs and usually presents as some combination of tingling, cramping discomfort, pain, numbness, or weakness in the lower back, calves, glutes, and thighs and is precipitated by walking and prolonged standing. However, the symptoms vary depending on the severity and cause of the condition.
The pectineus is the only adductor muscle that is innervated by the femoral nerve. The other adductor muscles are innervated by the obturator nerve [1] with the exception of a small part of the adductor magnus which is innervated by the tibial nerve. [4]
Weakness comes on slowly (over months to years) in an asymmetric manner and progresses steadily, leading to severe weakness and wasting of arm and leg muscles. IBM is more common in men than women. [10] Patients may become unable to perform activities of daily living and most require assistive devices within 5 to 10 years of symptom onset.