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The term "bendopnea" (meaning "bent" and "breath") was coined to be easily identifiable among patients and physicians. [ 3 ] Bendopnea should be distinguished from orthopnea (shortness of breath while lying down), trepopnea (shortness of breath while lying on one side), and platypnea (shortness of breath relieved by lying down and worsened when ...
Atrial fibrillation (AF, AFib or A-fib) is an abnormal heart rhythm (arrhythmia) characterized by rapid and irregular beating of the atrial chambers of the heart. [ 11 ] [ 12 ] It often begins as short periods of abnormal beating , which become longer or continuous over time. [ 4 ]
Lifetime risk of AFib has increased with 1 in 3 people at risk for the condition at some point during their lives. The CDC estimates that 12.1 million U.S. adults will have atrial fibrillation by ...
Certain medications that help control the heart rate might be given, or medications that reduce the likelihood of blood clot formation and therefore prevent stroke. Also, patients might receive an implantable cardiac pacemaker, which, by constantly pacing the atrium, can reduce the chance of an AF episode.
What causes Afib? Afib happens when cells in the upper chamber of the heart are triggered by something, causing an irregular heart rhythm in the upper chamber. ... The medications also come with ...
These drugs can be used to "rate control" a fast rhythm and make it physically tolerable for the patient. [citation needed] Some arrhythmias promote blood clotting within the heart and increase the risk of embolus and stroke. Anticoagulant medications such as warfarin and heparins, and anti-platelet drugs such as aspirin can reduce the risk of ...
The underlying causes of sudden cardiac arrest can result from cardiac and non-cardiac etiologies. The most common underlying causes are different, depending on the patient's age. Common cardiac causes include coronary artery disease, non-atherosclerotic coronary artery abnormalities, structural heart damage, and inherited arrhythmias. Common ...
It presents the drugs on two axes, instead of one, and is presented in tabular form. On the Y axis, each drug is listed, in roughly the Singh-Vaughan Williams order. On the X axis, the channels, receptors, pumps, and clinical effects are listed for each drug, with the results listed in a grid.
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