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Glaucoma is a group of eye diseases that can lead to damage of the optic nerve. The optic nerve transmits visual information from the eye to the brain. Glaucoma may cause vision loss if left untreated. It has been called the "silent thief of sight" because the loss of vision usually occurs slowly over a long period of time. [5]
Glaucoma is the leading cause for irreversible blindness globally. [11] Glaucoma usually starts with no symptoms to start losing peripheral vision, and if untreated, it can leads to a complete loss of peripheral vision, which is tunnel vision, and eventually, central vision will be affected leading to complete blindness.
Over many years, glaucoma has been defined by an intraocular pressure of more than 20 mm Hg. Incompatible with this (now obsolete) definition of glaucoma was the ever larger number of cases that have been reported in medical literature in the 1980s and 1990s who had the typical signs of glaucomatous damage, like optic nerve head excavation and thinning of the retinal nerve fiber layer, while ...
Central retinal artery occlusion: CRAO is characterized by painless, acute vision loss in one eye. [11] Central retinal vein occlusion: CRVO causes sudden, painless vision loss that can be mild to severe. [12] Branch retinal vein occlusion: sudden painless vision loss or visual field defect are the main symptom of BRVO. [13]
Because uveitic glaucoma is a progressive stage of anterior non infectious uveitis, uveitic glaucoma involves signs and symptoms of both glaucoma and uveitis.. Patients with acute non infectious anterior uveitis may experience the following symptoms: pain, blurry vision, headache, photophobia (discomfort or pain due to light exposure), or the observance of haloes around lights.
Secondary glaucoma is a collection of progressive optic nerve disorders associated with a rise in intraocular pressure (IOP) which results in the loss of vision. In clinical settings, it is defined as the occurrence of IOP above 21 mmHg requiring the prescription of IOP-managing drugs. [ 1 ]
The diurnal variation for normal eyes is between 3 and 6 mmHg and the variation may increase in glaucomatous eyes. During the night, intraocular pressure may not decrease [17] despite the slower production of aqueous humour. [18] Glaucoma patients' 24-hour IOP profiles may differ from those of healthy individuals. [19]
Ocular hypertension is treated with either medications (eye drops), surgery, or laser. Treatment, by lowering the intraocular pressure, may help decrease the risk of vision loss and damage to the eye from glaucoma. Treatment options include pressure-lowering 'antiglaucomatous' eye drops, surgery, and/or laser eye surgery. [4]
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