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In the longer term, the knee may not regain full movement, pain may persist and there is an increased likelihood of osteoarthritis of the knee. [7] If there is an associated open wound as in an open fracture , complications also include risk of infection, inability of the broken bones to reunite and osteonecrosis .
This complex is the major stabilizer of the medial knee. Injuries to the medial side of the knee are most commonly isolated to these ligaments. [1] [3] A thorough understanding of the anatomy and function of the medial knee structures, along with a detailed history and physical exam, are imperative to diagnosing and treating these injuries.
The knee anatomy provides insight into predicting why certain fracture patterns occur more often than others. The medial plateau is larger and significantly stronger than the lateral plateau. Also, there is a natural valgus or outward angulation alignment to the limb which coupled with the often valgus or outwardly angulating force on impact ...
When the damage increases and the chondral defect reaches the subchondral bone, the blood supply in the bone starts a healing process in the defect. Scar tissue made up of a type of cartilage called fibrocartilage is then formed. Although fibrocartilage is able to fill in articular cartilage defects, its structure is significantly different ...
In February 2008, Saris et al. published a large-scale study claiming that CCI results in better structural repair for symptomatic cartilage defects of the knee than microfracture surgery. According to the study, one year after treatment, the regenerated tissue associated with CCI is of better quality than that of microfracture surgery. [15]
External fixation is a surgical treatment wherein Kirschner pins and wires are inserted and affixed into bone and then exit the body to be attached to an external apparatus composed of rings and threaded rods — the Ilizarov apparatus, the Taylor Spatial Frame, and the Octopod External Fixator — which immobilises the damaged limb to facilitate healing. [1]
There doesn't seem to be any sign of hereditary factors at the moment, it is shown that Pellegrini-Stieda Syndrome is a result of a knee injury and ossification of the knee after a certain amount of time. When a full range of motion in the knee is attained, and the athlete does not feel any form of pain, they can resume their activities.
Originally described by Dr. Paul Segond in 1879 [6] [7] after a series of cadaveric experiments, the Segond fracture occurs in association with tears of the anterior cruciate ligament (ACL) (75–100%) and injury to the medial meniscus (66–75%), lateral capsular ligament (now known as the Anterolateral ligament, or ALL), as well as injury to the structures behind the knee.