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  2. PTEN (gene) - Wikipedia

    en.wikipedia.org/wiki/PTEN_(gene)

    Phosphatase and tensin homolog (PTEN) is a phosphatase in humans and is encoded by the PTEN gene. [6] Mutations of this gene are a step in the development of many cancers , specifically glioblastoma, lung cancer, breast cancer, and prostate cancer.

  3. Cancer Treatment Centers of America - Wikipedia

    en.wikipedia.org/wiki/Cancer_Treatment_Centers...

    Cancer Treatment Centers of America was the subject of a Federal Trade Commission (FTC) complaint in 1993 alleging that CTCA made false claims regarding the success rates of certain cancer treatments in marketing and promotional materials. Among other unsubstantiated claims, CTCA advertised that it was able to treat certain forms of cancer ...

  4. Bannayan–Riley–Ruvalcaba syndrome - Wikipedia

    en.wikipedia.org/wiki/Bannayan–Riley...

    The genetics of the Bannayan–Riley–Ruvalcaba syndrome is determined, in the majority of cases, via the PTEN gene which presents about 30 mutations in this condition. This gene which regulates cell growth, when not working properly can lead to hamartomas. PTEN chromosomal location is 10q23.31, while the molecular location is 87,863,438 to ...

  5. John Theurer Cancer Center - Wikipedia

    en.wikipedia.org/wiki/John_Theurer_Cancer_Center

    It is known for being the first site approved to use cell-based gene therapy to treat patients with certain types of large B-cell lymphoma who have not responded or relapsed after at least two other kinds of treatment; it initiated the first CAR T-cell therapy clinical trials and is a member of the NCI-approved Georgetown Lombardi Comprehensive ...

  6. PI3K/AKT/mTOR pathway - Wikipedia

    en.wikipedia.org/wiki/PI3K/AKT/mTOR_pathway

    The most common known aberrations include the PIK3CA gene mutation and the loss-of-function mutations or epigenetic silencing of PTEN. [12] The phosphoinositide 3-kinase (PI3K)/protein kinase B (Akt)/mammalian target of rapamycin (mTOR) pathway is activated in approximately 30–40% of BC cases.

  7. Phosphoinositide 3-kinase - Wikipedia

    en.wikipedia.org/wiki/Phosphoinositide_3-kinase

    Many of these mutations cause the kinase to be more active. It is the single most mutated kinase in glioblastoma , the most malignant primary brain tumor. [ 22 ] The PtdIns(3,4,5) P 3 phosphatase PTEN that antagonises PI3K signaling is absent from many tumours.

  8. The Cancer Genome Atlas - Wikipedia

    en.wikipedia.org/wiki/The_Cancer_Genome_Atlas

    The Cancer Genome Atlas (TCGA) is a project to catalogue the genomic alterations responsible for cancer using genome sequencing and bioinformatics. [1] [2] The overarching goal was to apply high-throughput genome analysis techniques to improve the ability to diagnose, treat, and prevent cancer through a better understanding of the genetic basis of the disease.

  9. Pseudogene - Wikipedia

    en.wikipedia.org/wiki/Pseudogene

    PTEN. The PTEN gene is a known tumor suppressor gene. The PTEN pseudogene, PTENP1 is a processed pseudogene that is very similar in its genetic sequence to the wild-type gene. However, PTENP1 has a missense mutation which eliminates the codon for the initiating methionine and thus prevents translation of the normal PTEN protein. [47]