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Benign prostatic hyperplasia (BPH), also called prostate enlargement, is a noncancerous increase in size of the prostate gland. [1] Symptoms may include frequent urination, trouble starting to urinate, weak stream, inability to urinate, or loss of bladder control. [1]
In the male the pubovesical ligament is parallel and medial to the puboprostatic ligament. The puboprostatic ligament is a thickening of the superior fascia of the pelvic diaphragm, that extends laterally from the prostate to the tendinous arch of the pelvic fascia and continues forward and medially from the tendinous arch to the pubis.
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The detrusor muscle is able to change its length. It can also contract for a long time whilst voiding, and it stays relaxed whilst the bladder is filling. [9] The wall of the urinary bladder is normally 3–5 mm thick. [10] When well distended, the wall is normally less than 3 mm.
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The female or male external sphincter muscle of urethra (sphincter urethrae): located in the deep perineal pouch, at the bladder's distal inferior end in females, and inferior to the prostate (at the level of the membranous urethra) in males. It is a secondary sphincter to control the flow of urine through the urethra.
Hydronephrosis can also result from the retrograde flow of urine from the bladder back into the kidneys (vesicoureteral reflux), which can be caused by some of the factors listed above as well as compression of the bladder outlet into the urethra by prostate enlargement or fecal impaction in the rectum (which sits immediately behind the ...
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