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Central nervous system fatigue is a key component in preventing peripheral muscle injury. [28] The brain has numerous receptors, such as osmoreceptors, to track dehydration, nutrition, and body temperature. With that information as well as peripheral muscle fatigue information, the brain can reduce the quantity of motor commands sent from the ...
Nootropics (/ n oʊ. ə ˈ t r oʊ p ɪ k s / noh-ə-TROHP-iks or / n oʊ. ə ˈ t r ɒ p ɪ k s / noh-ə-TROP-iks; [1] but not / n j uː ˈ t r oʊ p ɪ k s / new-TROHP-iks or / n j uː ˈ t r ɒ p ɪ k s / new-TROP-iks, [1] which are common mispronunciations [citation needed]), colloquially brain supplements, smart drugs and cognitive ...
Caffeine. Caffeine is a central nervous system (CNS) stimulant of the methylxanthine class and is the most commonly consumed psychoactive substance globally. [9][10] It is mainly used for its eugeroic (wakefulness promoter), ergogenic (physical performance enhancing), or nootropic (cognition improving) properties. [11][12] Caffeine acts by ...
Facilitate rapid recovery (by reducing central nervous system fatigue). Most effective in individuals with low to moderate baseline resilience to stress. Involve a complexity of biochemical processes. Decrease entropy of an organism (lowering oxygen consumption, body temperature, heart rate, etc).
Vitamin D and neurological disorders. Hypovitaminosis D is associated with several neuropsychiatric disorders including dementia, Parkinson's disease, multiple sclerosis, epilepsy, and schizophrenia. There are several proposed mechanisms by which hypovitaminosis D may impact these disorders. One of these mechanisms is through neuronal apoptosis ...
Neuroregeneration is the regrowth or repair of nervous tissues, cells or cell products. Neuroregenerative mechanisms may include generation of new neurons, glia, axons, myelin, or synapses. Neuroregeneration differs between the peripheral nervous system (PNS) and the central nervous system (CNS) by the functional mechanisms involved, especially ...
Cannabinoid receptor 1 (CB1), is a G protein-coupled cannabinoid receptor that in humans is encoded by the CNR1 gene. [5] And discovered, by determination and characterization in 1988, [6] and cloned in 1990 for the first time. [7][8][9] The human CB1 receptor is expressed in the peripheral nervous system and central nervous system. [5]
Megavitamin-B6 syndrome, also known as hypervitaminosis B6, vitamin B6 toxicity, and vitamin B6 excess, [a] is a medical condition characterized by adverse effects resulting from excessive intake of vitamin B 6. [1][2][22] Primarily affecting the nervous system, this syndrome manifests through symptoms such as peripheral sensory neuropathy ...
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