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It is usually resistant to a variety of antibiotics including penicillins, cephalosporins, quinolones, and aminoglycosides. Ampicillin and carbenicillin, which are penicillins, are an exception. It is variably susceptible to tetracyclines, chloramphenicol, trimethoprim-sulfamethoxazole, and colistin.
Peritoneal dialysis was first carried out in the 1920s; however, long-term use did not come into medical practice until the 1960s. [35] The timeline was 1923 – Georg Ganter performs the first peritoneal dialysis in a guinea pig and attempts the procedure in humans, without success. Hypertonic saline was used as the dialysate. [35] [36]
Spontaneous bacterial peritonitis (SBP) is a peculiar form of peritonitis occurring in the absence of an obvious source of contamination. It occurs in people with ascites, including children. Intra-peritoneal dialysis predisposes to peritoneal infection (sometimes named "primary peritonitis" in this context).
Spontaneous bacterial peritonitis (SBP) is the development of a bacterial infection in the peritoneum, despite the absence of an obvious source for the infection. [1] It is specifically an infection of the ascitic fluid – an increased volume of peritoneal fluid. [2] Ascites is most commonly a complication of cirrhosis of the liver. [1]
Another noteworthy case involves a 66-year-old woman with end-stage renal disease due to type 2 diabetes, who developed peritoneal dialysis-associated peritonitis attributed to Kytococcus sedentarius. This case highlights the potential for touch contamination in infections by Kytococcus sedentarius. [13]
Pseudomonas oryzihabitans, although an uncommon pathogen, is able to cause infections in individuals that usually have compromised immune systems.While most strains of this bacteria do not cause infections, the patients that acquire P. oryzihabitans most likely have an underlying disease, and it spreads while the patients are hospitalized. [5]
The following is a list of antibiotics. The highest division between antibiotics is bactericidal and bacteriostatic. Bactericidals kill bacteria directly, whereas bacteriostatics prevent them from dividing. However, these classifications are based on laboratory behavior.
D. acidovorans is an emergent opportunistic pathogen that demonstrates antibiotic resistance. [6] [13] The infection can cause bacteremia, [14] keratitis, [15] [16] pneumonia, [17] empyema, [18] otitis, [19] and peritonitis. [20] Known sources of infection include contaminated water [19] and catheters.
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