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  2. Neonatal cholestasis - Wikipedia

    en.wikipedia.org/wiki/Neonatal_cholestasis

    Neonatal cholestasis refers to elevated levels of conjugated bilirubin identified in newborn infants within the first few months of life. [1] Conjugated hyperbilirubinemia is clinically defined as >20% of total serum bilirubin or conjugated bilirubin concentration greater than 1.0 mg/dL regardless of total serum bilirubin concentration. [2]

  3. Hereditary hyperbilirubinemia - Wikipedia

    en.wikipedia.org/wiki/Hereditary_hyperbilirubinemia

    Hereditary hyperbilirubinemia refers to a group of conditions where levels of bilirubin, a byproduct of red blood cell metabolism, are elevated in the blood due to a genetic cause. [1] Various mutations of enzymes in the liver cells, which breakdown bilirubin, cause varying elevated levels of bilirubin in the blood. [2]

  4. Neonatal jaundice - Wikipedia

    en.wikipedia.org/wiki/Neonatal_jaundice

    [5] [6] Quick and accurate treatment of neonatal jaundice helps to reduce the risk of neonates developing kernicterus. [7] Infants with kernicterus may have a fever [8] or seizures. [9] High pitched crying is an effect of kernicterus. [citation needed] Exchange transfusions performed to lower high bilirubin levels are an aggressive treatment. [10]

  5. Crigler–Najjar syndrome - Wikipedia

    en.wikipedia.org/wiki/Crigler–Najjar_syndrome

    Crigler–Najjar syndrome is a rare inherited disorder affecting the metabolism of bilirubin, a chemical formed from the breakdown of the heme in red blood cells. The disorder results in a form of nonhemolytic jaundice, which results in high levels of unconjugated bilirubin and often leads to brain damage in infants.

  6. Rotor syndrome - Wikipedia

    en.wikipedia.org/wiki/Rotor_syndrome

    Rotor syndrome (also known as Rotor type hyperbilirubinemia) [2] is a rare cause of mixed direct (conjugated) and indirect (unconjugated) hyperbilirubinemia, relatively benign, autosomal recessive [3] bilirubin disorder characterized by non-hemolytic jaundice due to the chronic elevation of predominantly conjugated bilirubin.

  7. Vanishing bile duct syndrome - Wikipedia

    en.wikipedia.org/wiki/Vanishing_Bile_Duct_Syndrome

    The presentation is dependent upon the underlying cause. The course can be rapid or chronic. Fatigue; Anorexia; Abdominal pain; Weight loss; Pruritus; Hyperlipidemia; Malabsorption; Fat-soluble vitamin deficiencies; Elevated alkaline phosphatase; Elevated gamma-glutamyltransferase; Elevated conjugated bilirubin

  8. Hyperbilirubinemia in adults - Wikipedia

    en.wikipedia.org/wiki/Hyperbilirubinemia_in_adults

    The varied causes of hyperbilirubinemia are best understood from bilirubin metabolism. The total average daily production of bilirubin in humans is 4 mg/kg. 80% of which is derived from haemoglobin (Hb) produced by erythrocyte breakdown, with the remaining from heme proteins, like myoglobin and cytochrome, turnover. [5]

  9. Gilbert's syndrome - Wikipedia

    en.wikipedia.org/wiki/Gilbert's_syndrome

    People with GS predominantly have elevated unconjugated bilirubin, while conjugated bilirubin is usually within the normal range or is less than 20% of the total. Levels of bilirubin in GS patients are reported to be from 20 μM to 90 μM (1.2 to 5.3 mg/dl) [38] compared to the normal amount of < 20 μM. GS patients have a ratio of unconjugated ...